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Acute inhalation injury may result from frequent and widespread use of household cleaning agents and industrial gases (including chlorine and ammonia).The airways and lungs receive continuous first-pass exposure to non-toxic and irritant or toxic gases via inhalation.
Acute chlorine gas poisoning primarily affects the respiratory system, causing difficulty breathing, cough, irritation of the eyes, nose, and throat, and sometimes skin irritation. Higher exposures can lead to severe lung damage, such as toxic pneumonitis or pulmonary edema, with concentrations around 400 ppm and beyond
Smoke inhalation is the breathing in of harmful fumes (produced as by-products of combusting substances) through the respiratory tract. [1] This can cause smoke inhalation injury (subtype of acute inhalation injury) which is damage to the respiratory tract caused by chemical and/or heat exposure, as well as possible systemic toxicity after smoke inhalation.
The following tests help determine how severely the lungs are affected: Blood gases (measurement of how much oxygen and carbon dioxide are in your blood) CT scan of chest; Lung function studies (tests to measure breathing and how well the lungs are functioning) X-ray of the chest; Swallowing studies to check if stomach acid is the cause of ...
Respiratory arrest is a serious medical condition caused by apnea or respiratory dysfunction severe enough that it will not sustain the body (such as agonal breathing). Prolonged apnea refers to a patient who has stopped breathing for a long period of time. If the heart muscle contraction is intact, the condition is known as respiratory arrest.
Oxygen toxicity is a condition resulting from the harmful effects of breathing molecular oxygen (O 2) at increased partial pressures.Severe cases can result in cell damage and death, with effects most often seen in the central nervous system, lungs, and eyes.
Exposure to nitrogen dioxide causes ovulation-induced oxidative damage to the DNA of ovarian epithelium. [54] There is a growing body of literature on the pathological effects of ROS on female reproduction as evidenced by free-radical-induced birth defects, abortions, hydatidiform moles and pre-eclampsia.
The underlying mechanism involves diffuse injury to cells which form the barrier of the microscopic air sacs of the lungs, surfactant dysfunction, activation of the immune system, and dysfunction of the body's regulation of blood clotting. [5] In effect, ARDS impairs the lungs' ability to exchange oxygen and carbon dioxide. [1]
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