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Parapsoriasis refers to one of a group of skin disorders that are characterized primarily by their resemblance to psoriasis (red, scaly lesions), rather than by their underlying cause. Neoplasms can develop from parapsoriasis. [1] For example, it can develop into cutaneous T-cell lymphoma. The word "parapsoriasis" was formed in 1902. [2]
Large plaque parapsoriasis are skin lesions that may be included in the modern scheme of cutaneous conditions described as parapsoriasis. [2] These lesions, called plaques, may be irregularly round-shaped to oval and are 10 cm (4 in) or larger in diameter. [2] They can be very thin plaques that are asymptomatic or mildly pruritic.
Large plaque parapsoriasis consists of inflamed, oddly discolored (such as yellow or blue), web-patterned and scaling plaques on the skin, 10 cm (3.9 in) or larger in diameter. [5] When the condition of the skin encompassed by these plaques worsens and becomes atrophic, it is typically considered retiform parapsoriasis. [5]
Small plaque parapsoriasis characteristically occurs with skin lesions that are round, oval, discrete patches or thin plaques, mainly on the trunk. [2]: 452 [3]: 207 [4] Subtypes: Xanthoerythrodermia perstans is a distinct variant with lesions that are yellow in color. [1] [2]: 452
Acute guttate parapsoriasis, Acute parapsoriasis, Acute pityriasis lichenoides, Mucha–Habermann disease, Parapsoriasis acuta, Parapsoriasis lichenoides et varioliformis acuta, Parapsoriasis varioliformis [1]: 456 [2]: 736 )
Retiform parapsoriasis is a cutaneous condition, considered to be a type of large-plaque parapsoriasis. [1] It is characterized by widespread, ill-defined plaques on the skin, that have a net-like or zebra-striped pattern. [2] Skin atrophy, a wasting away of the cutaneous tissue, usually occurs within the area of these plaques. [1]
Psoriasis is a long-lasting, noncontagious autoimmune disease characterized by patches of abnormal skin. [4] [5] These areas are red, pink, or purple, dry, itchy, and scaly.[8] [3] Psoriasis varies in severity from small localized patches to complete body coverage. [3]
Psoriatic erythroderma can be congenital or secondary to an environmental trigger. [12] [13] [14] Environmental triggers that have been documented include sunburn, skin trauma, psychological stress, systemic illness, alcoholism, drug exposure, chemical exposure (e.g., topical tar, computed tomography contrast material), and the sudden cessation of medication.
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