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Ototoxicity is the property of being toxic to the ear (oto-), specifically the cochlea or auditory nerve and sometimes the vestibular system, for example, as a side effect of a drug. The effects of ototoxicity can be reversible and temporary, or irreversible and permanent. It has been recognized since the 19th century. [1]
The syndrome is notable not only for its impact on peripheral nerve function but also because of its generally, but not always, reversible nature upon cessation of vitamin B 6 intake. Usually, but not always, cases resolve within six months after stopping the vitamin B 6 supplementation, although some symptoms can intensify briefly after ...
Liver damage [5] Reserpine: Rauvolfia serpentina "Sedation, inability to complete tasks, mental depression, nasal congestion, increased gastric secretion and mild diarrhea" [3] Safrole: Sassafras albidum: Liver damage [3] Saw palmetto: Serenoa repens
Sensorineural hearing loss (SNHL) is a type of hearing loss in which the root cause lies in the inner ear, sensory organ (cochlea and associated structures), or the vestibulocochlear nerve (cranial nerve VIII). SNHL accounts for about 90% of reported hearing loss.
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Cochlear hydrops preferentially affects the apex of the cochlea where low-frequency sounds are interpreted. Due to the fluid imbalance in this area, parts of the cochlea are stretched or under more tension than usual, which can lead to distortions of sound, changes in pitch perception, or hearing loss, all usually in the low frequencies.
A neuropathy usually refers to a disease of the peripheral nerve or nerves, but the auditory nerve itself is not always affected in auditory neuropathy spectrum disorders. [2] Prevalence in the population is relatively unknown. Neonates with high risk factors for hearing loss have a prevalence of up to 40% (Vignesh, Jaya, & Muraleedharan 2016).
Vitamin D 2, or ergocalciferol, is obtained from D 2 supplements or fortified food sources. [3] These two forms of vitamin D are metabolized in the liver and stored as 25-hydroxyvitamin D. [ 4 ] Before biological use, the storage form must be converted into an active form.
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