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Posterior cortical atrophy (PCA), also called Benson's syndrome, is a rare form of dementia which is considered a visual variant or an atypical variant of Alzheimer's disease (AD). [1] [2] [3] The disease causes atrophy of the posterior part of the cerebral cortex, resulting in the progressive disruption of complex visual processing. [4]
Vascular dementia can be caused by ischemic or hemorrhagic infarcts affecting multiple brain areas, including the anterior cerebral artery territory, the parietal lobes, or the cingulate gyrus. [5] On rare occasion, infarcts in the hippocampus or thalamus are the cause of dementia. [12]
Causes Idiopathic , stroke , dementia Gerstmann syndrome is a neurological disorder that is characterized by a constellation of symptoms [ 1 ] that suggests the presence of a lesion usually near the junction of the temporal and parietal lobes at or near the angular gyrus .
Anosognosia is a condition in which a person with a disability is cognitively unaware of having it due to an underlying physical condition. Anosognosia results from physiological damage to brain structures, typically to the parietal lobe or a diffuse lesion on the fronto-temporal-parietal area in the right hemisphere, [1] [2] [3] and is thus a neuropsychiatric disorder.
Constructional apraxia is common after right parietal stroke and it continues after visuospatial symptoms have subsided. [5] Patients with posterior and parietal lobe lesions tend to have the most severe symptoms. [9] In Alzheimer's disease research, the AT8 antibody has proven to be an early indicator of tau protein pathology.
Animation. Parietal lobe (red) of left cerebral hemisphere. The parietal lobe is defined by three anatomical boundaries: The central sulcus separates the parietal lobe from the frontal lobe; the parieto-occipital sulcus separates the parietal and occipital lobes; the lateral sulcus (sylvian fissure) is the most lateral boundary, separating it from the temporal lobe; and the longitudinal ...
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