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General management principles for acute kidney injury include determination of volume status, fluid resuscitation with isotonic crystalloid, treatment of volume overload with diuretics,...
Acute kidney injury (AKI), previously called acute renal failure (ARF), denotes a sudden and often reversible reduction in kidney function, as measured by glomerular filtration rate (GFR).
Acute kidney injury (AKI) refers to an abrupt decrease in kidney function, resulting in the retention of urea and other nitrogenous waste products and in the dysregulation of extracellular volume and electrolytes.
Acute kidney injury is a rapid decrease in renal function over days to weeks, causing an accumulation of nitrogenous products in the blood (azotemia) with or without reduction in amount of urine output.
A sample of your blood may show fast-rising levels of urea and creatinine. This helps show how your kidneys are working. Urine output measures. Measuring how much urine you pass in 24 hours may help find the cause of your kidney failure.
Acute kidney injury is defined as an abrupt (within 48 hours) reduction in kidney func-tion based on an elevation in serum creati-nine level, a reduction in urine output, the need for renal...
Acute kidney injury (AKI) is a frequent diagnosis with an incidence that varies from 5.0% to 7.5% in hospitalized patients and that reaches up to 50–60% in critically ill patients [1, 2, 3, 4, 5, 6].
Acute kidney injury (AKI) is an abrupt and usually reversible decline in the glomerular filtration rate (GFR). This results in an elevation of serum blood urea nitrogen (BUN), creatinine, and other metabolic waste products that are normally excreted by the kidney.
Basic labs: Electrolytes (including Ca/Phos/Mg). Creatinine Kinase. Urinalysis (table below; if urinalysis suggests glomerulonephritis or acute interstitial nephritis, consult nephrology to review the urine microscopy). Additional labs: Relevant drug levels (e.g., vancomycin, aminoglycoside, cyclosporine, tacrolimus).
Summary. Acute. kidney injury. (AKI) is a sudden loss of renal function with a subsequent rise in. creatinine. and. blood urea nitrogen. ( BUN. ). It is most frequently caused by decreased renal. perfusion. ( prerenal. ) but may also be due to direct damage to the kidneys (intrarenal or intrinsic) or inadequate urine drainage (postrenal).