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As soman is closely related to compounds such as sarin, indications for a soman poisoning are relatively similar. One of the first observable signs of a soman poisoning is miosis . Some, but not all of the later indications are vomiting, extreme muscle pain and peripheral nervous system problems.
OPs are one of the most common causes of poisoning worldwide. [2] There are nearly 3 million poisonings per year resulting in two hundred thousand deaths. [2] [3] Around 15% of people who are poisoned die as a result. [2] Organophosphate poisoning has been reported at least since 1962. [7]
Developmental toxicity is any developmental malformation that is caused by the toxicity of a chemical or pathogen. It is the structural or functional alteration, reversible or irreversible, which interferes with homeostasis , normal growth , differentiation , development or behavior.
Tabun is toxic even in minute doses. The number and severity of symptoms which appear vary according to the amount of the agent absorbed and rate of entry of it into the body. Very small skin dosages sometimes cause local sweating and tremors accompanied with characteristically constricted pupils with few other effects.
Chronic solvent-induced encephalopathy (CSE) is a condition induced by long-term exposure to organic solvents, often—but not always—in the workplace, that lead to a wide variety of persisting sensorimotor polyneuropathies and neurobehavioral deficits even after solvent exposure has been removed.
Lead poisoning, also known as plumbism and saturnism, is a type of metal poisoning caused by lead in the body. [2] Symptoms may include abdominal pain, constipation, headaches, irritability, memory problems, infertility, and tingling in the hands and feet. [1]
Environmental toxicants and fetal development is the impact of different toxic substances from the environment on the development of the fetus.This article deals with potential adverse effects of environmental toxicants on the prenatal development of both the embryo or fetus, as well as pregnancy complications.
It causes death by respiratory paralysis resulting from disruption of the central nervous system. [2] It is a potent, noncompetitive antagonist of the gamma-aminobutyric acid (GABA) receptor. In humans, cicutoxin rapidly produces symptoms of nausea, emesis and abdominal pain, typically within 60 minutes of