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Global aphasia is a severe form of nonfluent aphasia, caused by damage to the left side of the brain, that affects [1] receptive and expressive language skills (needed for both written and oral language) as well as auditory and visual comprehension. [2]
In order to capitalize on neuroplasticity for treatment of all types of aphasia, timing, intensity, duration, and repetition of treatment should be taken into consideration. Research has found that aphasia treatment initiated during the earlier acute post-injury phase is more effective compared to treatment initiated in the chronic phase. [21]
Helm-Estabrooks is a researcher and speech-language pathologist (SLP) specializing in the design and application of widely used diagnostic. She developed, in collaboration with behavioral neurologist Martin Albert and Robert Sparks, a structured rehabilitation program for a type of nonfluent aphasia utilizing intoned phrases to facilitate speech and language production.
This is a mild form of aphasia as comprehension is not limited. Global aphasia [21] [22] is the most severe form of aphasia as there is difficulty with speech comprehension, as well as difficulty in responding in meaningful ways. This is caused by several brain injuries in more than one spot.
People also with anomic aphasia tend to know how to use an object, but rather can not name the aforementioned object. Any damage in or near the zone of language can result in anomic aphasia. Other forms of aphasia often transition into a syndrome of primarily anomic aphasia in the process of recovery. [3]
Mixed transcortical aphasia is characterized by severe speaking and comprehension impairment, but with preserved repetition. [6] People who suffer mixed transcortical aphasia struggle greatly to produce propositional language or to understand what is being said to them, yet they can repeat long, complex utterances or finish a song once they hear the first part.
Transcortical sensory aphasia is characterized as a fluent aphasia. Fluency is determined by direct qualitative observation of the patient’s speech to determine the length of spoken phrases, and is usually characterized by a normal or rapid rate; normal phrase length, rhythm, melody, and articulatory agility; and normal or paragrammatic speech. [5]
Progressive nonfluent aphasia (PNFA) is one of three clinical syndromes associated with frontotemporal lobar degeneration. PNFA has an insidious onset of language deficits over time as opposed to other stroke-based aphasias, which occur acutely following trauma to the brain.