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Feline odontoclastic resorption lesion (FORL) is a syndrome in cats characterized by resorption of the tooth by odontoclasts, cells similar to osteoclasts. FORL has also been called Feline tooth resorption ( TR ), neck lesion , cervical neck lesion , cervical line erosion , feline subgingival resorptive lesion , feline caries , or feline cavity .
They address various conditions such as jaw fractures, malocclusions of the teeth, oral cancer, periodontal disease, and unique veterinary conditions like feline odontoclastic resorptive lesions. Additionally, some animals have specialized dental workers like equine dental technicians, who perform routine dental work on horses.
External cervical resorption is a localised resorptive lesion in the cervical area of the tooth, below the epithelial attachment. It is distinguished from external inflammatory root resorption in that it rarely involves the pulp. When at least 3 teeth are affected, it is referred to as multiple idiopathic cervical root resorption.
Feline odontoclastic resorptive lesion; Feline spongiform encephalopathy; Polyneuropathy; Pyometra; Uterine unicornis a condition in which the female cat is missing a uterine horn. A rare discovery by veterinarians, the condition can be detected by x-ray or ultrasound prior to spaying if the patient has a family history of the medical condition.
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If a tooth is infected by dental caries there are two main treatment options. Teeth with dental caries get extracted in most veterinary clinics. A second option is specialized vets that can save teeth from an extraction. If there remains enough enamel the tooth can be repaired. In the case of decay to the tooth pulp, a root canal treatment is ...
These lesions can grow large because they apply pressure over the bone, causing resorption. The toxins released by the breakdown of granulation tissue are one of the common causes of bone resorption. There are two schools of thought regarding cyst expansion. [5] Complementary response to inflammation; Chemical reaction with Interleukin and ...
All forms of portosystemic shunts produce various neurological, gastrointestinal, and urinary symptoms. [3]Symptoms of congenital PSS usually appear by six months of age [4] and include failure to gain weight, vomiting, and signs of hepatic encephalopathy (a condition where toxins normally removed by the liver accumulate in the blood and impair the function of brain cells) such as seizures ...