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Structural model at atomic resolution of bacteriophage T4 [1] The structure of a typical myovirus bacteriophage Anatomy and infection cycle of bacteriophage T4.. A bacteriophage (/ b æ k ˈ t ɪər i oʊ f eɪ dʒ /), also known informally as a phage (/ ˈ f eɪ dʒ /), is a virus that infects and replicates within bacteria and archaea.
Transmission electron micrograph of multiple bacteriophages attached to a bacterial cell wall. A phageome is a community of bacteriophages and their metagenomes localized in a particular environment, similar to a microbiome. [1] [2] Phageome is a subcategory of virome, which is all of the viruses that are associated with a host or environment. [3]
Phages are also capable of interacting with species other than bacteria, e.g., such as phage-encoded exotoxin interaction with animals. [20] Phage therapy is an example of applied phage community ecology. [citation needed]
A prophage is a bacteriophage (often shortened to "phage") genome that is integrated into the circular bacterial chromosome or exists as an extrachromosomal plasmid within the bacterial cell. [1] Integration of prophages into the bacterial host is the characteristic step of the lysogenic cycle of temperate phages.
The dominant hosts for viruses in the ocean are marine microorganisms, such as bacteria. [14] Bacteriophages are harmless to plants and animals, and are essential to the regulation of marine and freshwater ecosystems [91] are important mortality agents of phytoplankton, the base of the foodchain in aquatic environments. [92]
The total sequence length ranges from 7,826 (in phage pMC8) to 509,170 bp (in phage pHS181) with the GC content of these phage being an average of 38.25%. [6] Within Bacillus phage there are 12 clusters (A-L), 28 subclusters, and 14 singletons. Clusters are groups of related genomes with the 12 clusters showing at least 50% homology between ...
Plant pathogens, organisms that cause infectious plant diseases, include fungi, oomycetes, bacteria, viruses, viroids, virus-like organisms, phytoplasmas, protozoa, nematodes and parasitic plants. [2] In most plant pathosystems, virulence depends on hydrolases and enzymes that degrade the cell wall.
Plants have evolved R genes (resistance genes) whose products mediate resistance to specific virus, bacteria, oomycete, fungus, nematode or insect strains. R gene products are proteins that allow recognition of specific pathogen effectors, either through direct binding or by recognition of the effector's alteration of a host protein. [ 6 ]