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It is a rare blood condition in which the β-pyrrole ring of the hemoglobin molecule has the ability to bind irreversibly to any substance containing a sulfur atom. [ 1 ] [ 2 ] When hydrogen sulfide (H 2 S) (or sulfide ions ) and ferrous ions combine in the heme of hemoglobin, the blood is thus incapable of transporting oxygen to the tissues.
Actinomycosis abscesses grow larger as the disease progresses, often over months. In severe cases, they may penetrate the surrounding bone and muscle to the skin, where they break open and leak large amounts of pus, which often contains characteristic granules filled with progeny bacteria. These granules are often called "sulfur granules" due ...
Over half of groundwater sources in India have fluoride above recommended levels. [9] Fluorosis can also occur as a result of volcanic activity. The 1783 eruption of the Laki volcano in Iceland is estimated to have killed about 22% of the Icelandic population, and 60% of livestock, as a result of fluorosis and sulfur dioxide gases. [10]
Symptoms: itching, bleaching or darkening of skin, burning sensations, trouble breathing, coughing blood and/or tissue necrosis: Complications: Depends on the severity: Causes: most common include: sulfuric acid, hydrochloric acid, sodium hydroxide, lime, silver nitrate, and greater than 5% hydrogen peroxide solutions. Treatment
Yokkaichi asthma (四日市ぜんそく, Yokkaichi zensoku) refers to cases of chronic obstructive pulmonary disease, chronic bronchitis, pulmonary emphysema, and bronchial asthma in humans and various environmental changes usually attributed to sulfur dioxide (SO 2) emissions which appeared as smog over the city of Yokkaichi in Mie Prefecture, Japan between 1960 and 1972, though other SO x ...
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Attacks are most common in young adult women and are rare before puberty or after menopause. [8] Severe acute attacks may require hospitalization. Patients usually experience symptoms in attacks that last from several hours to a few days. Between attacks, patients are asymptomatic. [citation needed]
Phenol sulfur transferase deficiency, in short PST deficiency, is the lack or the reduced activity of the functional enzyme phenol sulfur transferase, which is crucial in the detoxification of mainly phenolic compounds by catalysing the sulfate conjugation of the hydroxyl groups in the toxic phenolic compounds to result in more hydrophilic forms for more efficient excretion.