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Asherman's syndrome (AS) is an acquired uterine condition that occurs when scar tissue forms inside the uterus and/or the cervix. [1] It is characterized by variable scarring inside the uterine cavity, where in many cases the front and back walls of the uterus stick to one another.
Severe cutaneous adverse reactions (SCARs) are a group of potentially lethal adverse drug reactions that involve the skin and mucous membranes of various body openings such as the eyes, ears, and inside the nose, mouth, and lips. In more severe cases, SCARs also involves serious damage to internal organs. SCARs includes five syndromes:
Asherman has published dozens of medical articles. Among other things, he described a syndrome that bears his name, Asherman's Syndrome, which includes scarring and infection in the uterus due to curettage, which was first described by a German doctor ( Heinrich Fritsch ) in 1894 but was characterized following two articles by Asherman in 1948 ...
An important risk factor for placenta accreta is placenta previa in the presence of a uterine scar. Placenta previa is an independent risk factor for placenta accreta. Additional reported risk factors for placenta accreta include maternal age and multiparity, other prior uterine surgery, prior uterine curettage, uterine irradiation, endometrial ablation, Asherman syndrome, uterine leiomyomata ...
Vaginal stenosis is an abnormal condition in which the vagina becomes narrower and shorter due to the formation of fibrous tissue. [1] [2] Vaginal stenosis can contribute to sexual dysfunction, dyspareunia and make pelvic exams difficult and painful. [1]
Vesicouterine fistulas occur most commonly after lower segment caesarean sections (about 83-93% of cases). [2] The possible mechanisms by which vesicouterine fistulas occur following caesarean sections include undetected bladder injury during caesarean section, inadvertent placement of a suture through the bladder during the repair of the uterus and abnormal blood vessel connections following ...
Studies of patients with lipodermatosclerosis have demonstrated significantly decreased concentrations of cutaneous oxygen associated with decreased capillary density. Capillaries are virtually absent in areas of fibrotic scars, leading to a condition known as atrophie blanche or livedoid vasculopathy. [8]
The goal of treatment with immunosuppressive agents such as corticosteroids is to decrease lung inflammation and subsequent scarring. Responses to treatment vary. Those whose conditions improve with immunosuppressive treatment probably do not have idiopathic pulmonary fibrosis, for idiopathic pulmonary fibrosis has no significant treatment or cure.