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Visual field-tubular vision Visual field-central scotoma. Lesions involving the whole optic nerve cause complete blindness on the affected side, that means damage at the right optic nerve causes complete loss of vision in the right eye. [3] Optic neuritis involving external fibers of the optic nerve causes tunnel vision. [4]
Thus the emerging perspective within neuropsychology and neurophysiology is that, whilst a two-systems framework was a necessary advance to stimulate study of the highly complex and differentiated functions of the two neural pathways; the reality is more likely to involve considerable interaction between vision-for-action and vision-for-perception.
More recently, robust Muller glia proliferation and subsequent neuronal differentiation has been seen using the alpha 7 nAChR agonist, PNU-282987. [10] More information on the signaling pathways involved is required before Muller glia mediated regeneration will be a viable treatment method for restoring vision in mammalian retinas.
A potential solution for the treatment of rare inherited retinal diseases is to develop gene therapies that target shared pathways in a manner similar to gene therapies for acquired retinal diseases.
Schematic diagram of pupillary light reflex neural pathway. Left direct light reflex involves neural segments 1, 5, and 7. Segment 1 is the afferent limb, which includes the retina and optic nerve. Segments 5 and 7 form the efferent limb. Left consensual light reflex involves neural segments 2, 4, and 7. Segment 2 is the afferent limb.
A neural pathway connects one part of the nervous system to another using bundles of axons called tracts. The optic tract that extends from the optic nerve is an example of a neural pathway because it connects the eye to the brain; additional pathways within the brain connect to the visual cortex.
The visual system is the physiological basis of visual perception (the ability to detect and process light).The system detects, transduces and interprets information concerning light within the visible range to construct an image and build a mental model of the surrounding environment.
The groups of Alexander Gottschalk and Georg Nagel made the first ChR2 mutant (H134R) and were first to use channelrhodopsin-2 for controlling neuronal activity in an intact animal, showing that motor patterns in the roundworm C. elegans could be evoked by light stimulation of genetically selected neural circuits (published in December 2005). [37]
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