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Dentin hypersensitivity is a relatively common condition. [4] [3] Due to differences in populations studied and methods of detection, the reported incidence ranges from 4-74%. [3] Dentists may under-report dentin hypersensitivity due to difficulty in diagnosing and managing the condition. [4]
Epidemiological surveys have shown that dentine hypersensitivity arises when the dentinal tubules are both exposed and patent. It was proposed that if the hydrodynamic fluid flow was responsible for hypersensitivity, then there must be higher numbers of dentinal tubules exposed at the surface of the root and patent to the dental pulp.
Hypersensitivity is most commonly caused by a lack of insulation from the triggers in the mouth due to gingival recession (receding gums) exposing the roots of the teeth, although it can occur after scaling and root planing or dental bleaching, or as a result of erosion. [19] The pulp of the tooth remains normal and healthy in dentin ...
If the attrition is severe, the enamel can be completely worn away leaving underlying dentin exposed, resulting in an increased risk of dental caries and dentin hypersensitivity. It is best to identify pathological attrition at an early stage to prevent unnecessary loss of tooth structure as enamel does not regenerate.
For early lesions of inflammatory papillary hyperplasia, cessation of denture use for 2 to 4 weeks may allow the lesion to completely subside. This may be aided by use of topical antibiotic or antifungal therapies. [25] Small lesions are also typically treated with mouthrinses such as chlorhexidine mouthrinse at 0.12% or antifungal mouthrinse ...
Maturation of dentin or mineralization of predentin occurs soon after its apposition, which takes place two phases: primary and secondary. Initially, the calcium hydroxyapatite crystals form as globules, or calcospherules, in the collagen fibers of the predentin, which allows for both the expansion and fusion during the primary mineralization ...
This change to hyperkeratinization commonly occurs on the usually nonkeratinized buccal mucosa when the linea alba forms, a white ridge of calloused tissue that extends horizontally at the level where the maxillary and mandibular teeth come together and occlude. Histologically, an excess amount of keratin is noted on the surface of the tissue ...
In some species, this is a normal process that occurs during the formation or maintenance of the dentition. [1] By contrast, in humans tooth ankylosis is pathological, whereby a fusion between alveolar bone and the cementum of a tooth occurs. In humans, this is a rare phenomenon in deciduous dentition and even more uncommon in permanent teeth.