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Adie syndrome, also known as Holmes–Adie syndrome, is a neurological disorder characterized by a tonically dilated pupil that reacts slowly to light but shows a more definite response to accommodation (i.e., light-near dissociation). [1] It is frequently seen in females with absent knee or ankle jerks and impaired sweating.
Adie syndrome [4] is tonic pupil plus absent deep tendon reflexes. Adie syndrome is a fairly common, benign, idiopathic neuropathy that selectively affects the ciliary ganglion and the spinal cord neurons involved in deep tendon reflex arcs. It usually develops in middle age, although it can occur in children. A variant of Adie syndrome, Ross ...
A relative afferent pupillary defect (RAPD), also known as a Marcus Gunn pupil (after Robert Marcus Gunn), is a medical sign observed during the swinging-flashlight test [1] whereupon the patient's pupils excessively dilate when a bright light is swung from the unaffected eye to the affected eye. The affected eye still senses the light and ...
Adie's tonic pupil is usually associated with a benign peripheral neuropathy (Adie syndrome), not with syphilis. [6] When penicillin became widely available in the 1940s, the prevalence of AR pupils (which develop only after decades of untreated infection) decreased dramatically. AR pupils are now quite rare.
Adie tonic pupil: Tonic pupil is usually an isolated benign entity, presenting in young women. It may be associated with loss of deep tendon reflex (Adie's syndrome). Tonic pupil is characterized by delayed dilation of the iris, especially after near stimulus, segmental iris constriction, and sensitivity of pupil to a weak solution of pilocarpine.
Adie and his colleague James Collier wrote the neurology chapter in Price's Textbook of the Practice of Medicine which is considered to be the first account of neurology in a general textbook. [6] Adie also published articles on pupillary abnormalities, [7] "forced grasping and groping" in frontal lobe disorders, [8] and narcolepsy. [9]
Adie pupil: William John Adie: neurology: ciliary nerve damage: dilated pupil, poorly reactive but with normal near accommodation Adson's sign: Alfred Washington Adson: vascular surgery: thoracic outlet syndrome: obliteration of radial pulse with manoeuvres Alexander's law: Gustav Alexander: neurology, neurosurgery, ENT: vestibular lesions
Ross' syndrome consists of Adie's syndrome (myotonic pupils and absent deep tendon reflexes) plus segmental anhidrosis (typically associated with compensatory hyperhidrosis). [ 1 ] It was characterized in 1958 [ 2 ] [ 3 ] by A.T. Ross. [ 4 ]