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Recurrent or episodic rhabdomyolysis is commonly due to intrinsic muscle enzyme deficiencies, which are usually inherited and often appear during childhood. [10] [13] Many structural muscle diseases feature episodes of rhabdomyolysis that are triggered by exercise, general anesthesia or any of the other causes of rhabdomyolysis listed above. [10]
A renal diet is a diet aimed at keeping levels of fluids, electrolytes, and minerals balanced in the body in individuals with chronic kidney disease or who are on dialysis. Dietary changes may include the restriction of fluid intake, protein, and electrolytes including sodium, phosphorus, and potassium. [1]
A 2017 review indicated that a high-protein diet (superior to 1.5 g per kilogram of ideal body weight per day) may contribute to life-long risk of kidney damage, including progression of or potentially new onset chronic kidney disease.
Over time, high blood pressure can cause damage to the arteries that can lead to health conditions including stroke, heart disease, kidney problems and dementia. There are multiple risk factors ...
A low-protein diet is used as a therapy for inherited metabolic disorders, such as phenylketonuria and homocystinuria, and can also be used to treat kidney or liver disease. Low protein consumption appears to reduce the risk of bone breakage presumably through changes in calcium homeostasis. [1]
There is also an increase in the liver synthesis of cholesterol. Second, lipid catabolism is decreased due to lower levels of lipoprotein lipase, the main enzyme involved in lipoprotein breakdown. [13] Cofactors, such as apolipoprotein C2, may also be lost by increased filtration of proteins.
Magnesium is a co-factor for enzymes that help create ATP, the body's primary energy source, Derocha says. ... is associated with a reduced risk for heart disease and lower blood pressure ...
Protein toxicity is the effect of the buildup of protein metabolic waste compounds, like urea, uric acid, ammonia, and creatinine.Protein toxicity has many causes, including urea cycle disorders, genetic mutations, excessive protein intake, and insufficient kidney function, such as chronic kidney disease and acute kidney injury.