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Purpura, arthritis, and abdominal pain are known as the "classic triad" of Henoch–Schönlein purpura. [5] Purpura occur in all cases, joint pains and arthritis in 80%, and abdominal pain in 62%. Some include gastrointestinal hemorrhage as a fourth criterion; this occurs in 33% of cases, sometimes, but not necessarily always, due to ...
Cryofibrinogenemia refers to a condition classified as a fibrinogen disorder in which a person's blood plasma is allowed to cool substantially (i.e. from its normal temperature of 37 °C to the near-freezing temperature of 4 °C), causing the (reversible) precipitation of a complex containing fibrinogen, fibrin, fibronectin, and, occasionally, small amounts of fibrin split products, albumin ...
Schönlein described purpura rheumatica (Schönlein's disease) an allergic non-thrombopenic purpura rash that became known as Henoch–Schönlein purpura, though now known as IgA vasculitis. [3] [4] He also discovered the parasitic cause of ringworm or favus (Trichophyton schönleinii). [5]
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Immune complex glomerulonephritis, as seen in Henoch-Schönlein purpura; this is an example of IgA involvement in a nephropathy. The reaction can take hours, days, or even weeks to develop, depending on whether or not there is immunological memory of the precipitating antigen. Typically, clinical features emerge a week following initial antigen ...
Eduard Heinrich Henoch (June 16, 1820 Berlin – August 26, 1910) was a German physician. He taught at the Berlin University (1868–1894). Henoch was of Jewish descent, and was the nephew of Moritz Heinrich Romberg .
Purpura (/ ˈ p ɜːr p jʊər ə / [1]) is a condition of red or purple discolored spots on the skin that do not blanch on applying pressure. The spots are caused by bleeding underneath the skin secondary to platelet disorders, vascular disorders, coagulation disorders, or other causes. [ 2 ]
Serum sickness, Henoch-Schonlein Purpura Type IV Delayed-type T-lymphocytes Activated T-cells produce cytokines causing inflammation leading to tissue destruction Drug reaction with eosinophilia and systemic symptoms (i.e. DRESS syndrome or DIHS), Stevens–Johnson syndrome, toxic epidermal necrolysis, acute generalized exanthematous pustulosis