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Wellens' syndrome is an electrocardiographic manifestation of critical proximal left anterior descending (LAD) coronary artery stenosis in people with unstable angina. Originally thought of as two separate types, A and B, it is now considered an evolving wave form, initially of biphasic T wave inversions and later becoming symmetrical, often ...
Henrick Joan Joost (Hein J. J. ) Wellens, M.D., (1935–2020) was a Dutch cardiologist who is considered one of the founding fathers of clinical cardiac electrophysiology - a discipline which enables patients with cardiac arrhythmias to have catheter electrode mapping and ablation.
Wellens' sign or warning: Hein Wellens: cardiology: severe stenosis of LAD: characteristic ekg changes Wernicke encephalopathy: Carl Wernicke: neurology, psychiatry: thiamine deficiency: neurological symptoms caused by biochemical lesions of the central nervous system after exhaustion of B-vitamin reserves, in particular thiamine: Wernicke ...
Mark E. Josephson (1943-2017) was an American cardiologist and writer, who was in the 1970s one of the American pioneers of the medical cardiology subspecialty of cardiac electrophysiology.
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At the beginning of the 20th century, local Lutheran church leaders in Fort Wayne felt an urgent need for an additional hospital in the city. Led by Reverend Philip Wambsganss, they raised funds from the surrounding area, in 1904, the 25-bed Lutheran Hospital opened. [1]
Hein Wellens (1935–2020), Dutch cardiologist; Jacob Thomas Jozef Wellens (1726-1784), Roman Catholic bishop of Antwerp, Belgium; Jessie Wellens part of PrankvsPrank; Johan Wellens (born 1956), Belgian cyclist; Leo Wellens (born 1959), Belgian cyclist; Paul Wellens (born 1980), English rugby league footballer; Paul Wellens (cyclist) (born 1952 ...
He was a consultant in cardiology at Brigham Hospital until his death, and affiliated with six other hospitals in the United States. [7] Levine was a pioneer in the treatment of coronary thrombosis. He was the second American physician to diagnose the condition, which he detailed in his book, Clinical Heart Disease (1936). [8]