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Aphasia, also known as dysphasia, [a] is an impairment in a person’s ability to comprehend or formulate language because of damage to specific brain regions. [2] The major causes are stroke and head trauma; prevalence is hard to determine, but aphasia due to stroke is estimated to be 0.1–0.4% in developed countries. [3]
TMoA, or any other type of aphasia, is identified and diagnosed through the screening and assessment process. Screening can be conducted by an SLP or other professional when there is a suspected aphasia. [8] The screening does not diagnose aphasia, rather it points to the need for a further comprehensive assessment.
Landau–Kleffner syndrome (LKS), also called infantile acquired aphasia, acquired epileptic aphasia, [1] or aphasia with convulsive disorder, is a rare neurological syndrome that develops during childhood. [2] It is named after William Landau and Frank Kleffner, who characterized it in 1957 with a diagnosis of six children. [3] [4]
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Transcortical sensory aphasia is characterized as a fluent aphasia. Fluency is determined by direct qualitative observation of the patient’s speech to determine the length of spoken phrases, and is usually characterized by a normal or rapid rate; normal phrase length, rhythm, melody, and articulatory agility; and normal or paragrammatic speech. [5]
In neuropathy, primary progressive aphasia (PPA) [1] is a type of neurological syndrome in which language capabilities slowly and progressively become impaired. As with other types of aphasia, the symptoms that accompany PPA depend on what parts of the brain's left hemisphere are significantly damaged. However, unlike most other aphasias, PPA ...
[3] [7] Expressive aphasia differs from dysarthria, which is typified by a patient's inability to properly move the muscles of the tongue and mouth to produce speech. Expressive aphasia also differs from apraxia of speech, which is a motor disorder characterized by an inability to create and sequence motor plans for conscious speech. [8]
Logopenic progressive aphasia (LPA) is a variant of primary progressive aphasia. [1] It is defined clinically by impairments in naming and sentence repetition. [ 2 ] It is similar to conduction aphasia and is associated with atrophy to the left posterior temporal cortex and inferior parietal lobule .