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Coumaphos is a nonvolatile, fat-soluble phosphorothioate with ectoparasiticide properties: it kills insects and mites. It is well known by a variety of brand names as a dip or wash, used on farm and domestic animals to control ticks, mites, flies and fleas.
Specific treatments for acute pesticide poisoning are often dependent on the pesticide or class of pesticide responsible for the poisoning. However, there are basic management techniques that are applicable to most acute poisonings, including skin decontamination, airway protection, gastrointestinal decontamination, and seizure treatment.
Coumaphos [30] Checkmite Organophosphate: This is an insecticide that is used inside the beehive to combat varroa mites and small hive beetles, which are parasites of the honey bee. Overdoses can lead to bee poisoning. Relatively nontoxic Demeton: Systox Organophosphate <2 hours Highly toxic Demeton-S-methyl [31] Meta-systox Organophosphate
Poisoning is the harmful effect which occurs when toxic substances are introduced into the body. [1] The term "poisoning" is a derivative of poison, a term describing any chemical substance that may harm or kill a living organism upon ingestion. [2] Poisoning can be brought on by swallowing, inhaling, injecting or absorbing toxins through the skin.
The primary toxicological endpoint of concern to the EPA is cholinesterase inhibition; a common toxic effect of organophosphate poisoning. There is not a lot of data on the effect of phosmet in humans, but in rats the chemical was found to have an LD50 of 113 to 160 mg/kg through oral exposure, and an LD50 of 3160 to 4640 mg/kg through skin ...
The effects of exposure may be delayed but a high level could be fatal. Medical observation is indicated [ 24 ] Tests involving acute exposure of rats, mice, and rabbits have demonstrated that dichlorvos has high to extremely acute toxicity from oral or dermal exposure and extremely acute toxicity from inhalation [ 25 ]
Poisoning can occur if pyrethroid containing flea treatment products, which are intended for dogs, are used on cats. The livers of cats detoxify pyrethroids via glucuronidation more poorly than dogs, which is the cause of this difference. [ 15 ]
Azinphos-methyl is a neurotoxin derived from nerve agents developed during World War II. [4] It was first registered in the US in 1959 as an insecticide and is also used as active ingredient in organophosphate (OP) pesticides. [6]