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Global aphasia typically results from an occlusion to the trunk of the middle cerebral artery (MCA), [2] which affects a large portion of the perisylvian region of the left cortex. [7] Global aphasia is usually a result of a thrombotic stroke, which occurs when a blood clot forms in the brain's blood vessels.
Survivors with global aphasia may have great difficulty understanding and forming words and sentences, and generally experience a great deal of difficulty when trying to communicate. [2] With considerable speech therapy rehabilitation, global aphasia may progress into expressive aphasia or receptive aphasia.
The Boston Diagnostic Aphasia Examination provides a comprehensive exploration of a range of communicative abilities. Its results are used to classify patient's language profiles into one of the localization based classifications of aphasia: Broca's, Wernicke's, anomic, conduction, transcortical, transcortical motor, transcortical sensory, and global aphasia syndromes, although the test does ...
New research in aphasia treatment is showing the benefit of the Life Participation Approach to Aphasia (LPAA) in which goals are written based on the skills needed by the individual patient to participate in specific real-life situations (i.e. communicating effectively with nurses or gaining employment). [11]
Lesions in the superior temporal gyrus (STG) produce a more persistent global aphasia, which is associated with poor aphasia recovery. [19] [20] Patient specific factors relate to the patient's age of onset, education level and motivation for recovery. Younger patients have been reported to demonstrate a higher recovery rate than older patients ...
There is currently no standardized treatment for Wernicke's Aphasia, meaning treatment varies from patient to patient depending on the severity of the lesion and the resulting deficits. In some patients, the first step of action is to attempt to treat the possible causes for the aphasia, such as removing a brain tumor, or treating a nervous ...
Transcortical sensory aphasia is characterized as a fluent aphasia. Fluency is determined by direct qualitative observation of the patient’s speech to determine the length of spoken phrases, and is usually characterized by a normal or rapid rate; normal phrase length, rhythm, melody, and articulatory agility; and normal or paragrammatic speech. [5]
Progressive nonfluent aphasia (PNFA) is one of three clinical syndromes associated with frontotemporal lobar degeneration. PNFA has an insidious onset of language deficits over time as opposed to other stroke-based aphasias, which occur acutely following trauma to the brain.
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