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Cerebellar stroke syndrome is a condition in which the circulation to the cerebellum is impaired due to a lesion of the superior cerebellar artery, anterior inferior cerebellar artery or the posterior inferior cerebellar artery. [1] Cardinal signs include vertigo, headache, vomiting, and ataxia. [2]
Lateral medullary syndrome is the most common form of posterior ischemic stroke syndrome. It is estimated that there are around 600,000 new cases of this syndrome in the United States alone. [5] [outdated statistic] Those at the overall highest risk for lateral medullary syndrome are men at an average age of 55.06.
An estimated 15% of preterm infants who survive develop cerebral palsy and 27% of the infants who survive experience moderate to severe neurosensory deficits by the time they reach 18–24 months old. [7] Prognosis is very poor when IVH results from intracerebral hemorrhage related to high blood pressure and is even worse when hydrocephalus ...
The posterior inferior cerebellar artery (PICA) is the largest branch of the vertebral artery. It is one of the three main arteries that supply blood to the cerebellum , a part of the brain . Blockage of the posterior inferior cerebellar artery can result in a type of stroke called lateral medullary syndrome .
Medial medullary syndrome, also known as inferior alternating syndrome, hypoglossal alternating hemiplegia, lower alternating hemiplegia, [1] or Dejerine syndrome, [2] is a type of alternating hemiplegia characterized by a set of clinical features resulting from occlusion of the anterior spinal artery.
Intracerebral hemorrhage (ICH), also known as hemorrhagic stroke, is a sudden bleeding into the tissues of the brain (i.e. the parenchyma), into its ventricles, or into both. [3] [4] [1] An ICH is a type of bleeding within the skull and one kind of stroke (ischemic stroke being the other).
Occlusion of AICA is considered rare, but generally results in a lateral pontine syndrome, also known as AICA syndrome.The symptoms include sudden onset of vertigo, vomiting, nystagmus, dysarthria, falling to the side of the lesion (due to damage to vestibular nuclei), and a variety of same-side features including hemiataxia, loss of all types of sensation of the face (due to damage to the ...
Benedikt syndrome is caused by a lesion (infarction, hemorrhage, tumor, or tuberculosis) in the tegmentum of the midbrain and cerebellum. Specifically, the median zone is impaired. It can result from occlusion of the posterior cerebral artery [1] or paramedian penetrating branches of the basilar artery. [2]