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Human brain in the coronal orientation. Amygdalae are shown in dark red. The amygdala (/ ə ˈ m ɪ ɡ d ə l ə /; pl.: amygdalae / ə ˈ m ɪ ɡ d ə l i,-l aɪ / or amygdalas; also corpus amygdaloideum; Latin from Greek, ἀμυγδαλή, amygdalē, 'almond', 'tonsil' [1]) is a paired nuclear complex present in the cerebral hemispheres of vertebrates.
The amygdala connects to the thalamus both through the amygdalofugal pathway and through a direct connection to the dorsomedial nucleus of the thalamus. [13] After leaving the substantia innominata , the ventral amygdalofugal pathway continues on a medial path to enter the septal region, the lateral preoptic area , the hypothalamus , and the ...
The basolateral amygdala and nucleus accumbens shell together mediate specific Pavlovian-instrumental transfer, a phenomenon in which a classically conditioned stimulus modifies operant behavior. [7] [8] One of the main functions of the basolateral complex is to stimulate the fear response. The fear system is intended to avoid pain or injury.
The uncinate fasciculus is a white matter association tract in the human brain that connects parts of the limbic system such as the temporal pole, anterior parahippocampus, and amygdala in the temporal lobe with inferior portions of the frontal lobe such as the orbitofrontal cortex.
Sleep is an essential requirement for the body and brain and allows the closing down and resting of the body's systems. There are also findings that suggest that the daily build-up of toxins in the brain are removed during sleep. [96] Whilst awake the brain consumes a fifth of the body's total energy needs.
The ventromedial prefrontal cortex (vmPFC) is a part of the prefrontal cortex in the mammalian brain.The ventral medial prefrontal is located in the frontal lobe at the bottom of the cerebral hemispheres and is implicated in the processing of risk and fear, as it is critical in the regulation of amygdala activity in humans. [2]
S.M., sometimes referred to as SM-046, is an American woman with a peculiar type of brain damage that physiologically reduces her ability to feel fear.First described by scientists in 1994, [1] she has had exclusive and complete bilateral amygdala destruction since late childhood as a consequence of Urbach–Wiethe disease.
The extended amygdala is a macrostructure in the brain that is involved in reward cognition and defined by connectivity and neurochemical staining. [1] [2] It includes the central medial amygdala, sublenticular substantia innominata, and the bed nucleus of the stria terminalis. [1] The boundaries are indistinct in Nissl stained sections. [2]