Search results
Results from the WOW.Com Content Network
Cerebellar abiotrophy (CA), also called cerebellar cortical abiotrophy (CCA), is a genetic neurological disease in animals, best known to affect certain breeds of horses, dogs and cats. It can also develop in humans. It develops when the neurons known as Purkinje cells, located in the cerebellum of the brain, begin to die off. These cells ...
1860 engraving depicting the performing horse Marocco. A significant portion of medieval technical literature consists of treatises on veterinary care. [S 11] Arab and Muslim scholars made notable contributions to the knowledge of equine medicine, education, [5] and training, in part due to the contributions of the translator Ibn Akhî Hizâm, who wrote around 895, [6] and Ibn al-Awam, who ...
The human brain contains 86 billion neurons, with 16 billion neurons in the cerebral cortex. [ 2 ] [ 1 ] Neuron counts constitute an important source of insight on the topic of neuroscience and intelligence : the question of how the evolution of a set of components and parameters (~10 11 neurons, ~10 14 synapses) of a complex system leads to ...
In 2003, G. Fleissner and colleagues found iron-based receptors in the upper beaks of homing pigeons, both seemingly connected to the animal's trigeminal nerve. Research took a different direction in 2000, however, when Thorsten Ritz and colleagues suggested that a photoreceptor protein in the eye, cryptochrome , was a magnetoreceptor, working ...
2. Hollow Food Puzzles. As the name implies, these food puzzles are hollow, as they are meant to be filled with food. Their shape makes it challenging for dogs to extract the food, keeping them ...
Neospora hughesi is an obligate protozoan apicomplexan parasite that causes myelitis and equine protozoal myeloencephalitis (EPM) in horses, and has only been documented in North America. [1] EPM is a neurological disease from lesions in the spinal cord, brain stem, or brain from parasites such as N. hughesi or Sarcocystis neurona. [2]
Horses cannot pass the disease among themselves; that is, one horse cannot contract the disease from another infected horse. The horse is a dead-end, or aberrant, host of the parasite. Although all horses are believed to be susceptible to EPM the disease is usually found in younger horses typically around three to six years of age. [2]
In 2014, a whole genome study of the DNA differences between wolves and dogs found that dogs did not show a reduced fear response; they showed greater synaptic plasticity. Synaptic plasticity is widely believed to be the cellular correlate of learning and memory, and this change may have altered the learning and memory abilities of dogs. [16]