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Molybdenum cofactor deficiency is a rare human disease in which the absence of molybdopterin – and consequently its molybdenum complex, commonly called molybdenum cofactor – leads to accumulation of toxic levels of sulphite and neurological damage.
The agriculture industry uses 500 tons per year as a fertilizer. In particular, its use has been suggested for treatment of whiptail in broccoli and cauliflower in molybdenum-deficient soils. [4] [5] However, care must be taken because at a level of 0.3 ppm sodium molybdate can cause copper deficiencies in animals, particularly cattle. [3]
Molybdenum is an essential element in most organisms; a 2008 research paper speculated that a scarcity of molybdenum in the Earth's early oceans may have strongly influenced the evolution of eukaryotic life (which includes all plants and animals). [1] At least 50 molybdenum-containing enzymes have been identified, mostly in bacteria.
Molybdenum: When molybdenum is inhaled from coal ash dust, discomfort of the nose, throat, skin and eye can occur. [14] As a result, short-term molybdenum exposure can cause an increase of wheezing and coughing. [14] Furthermore, chronic exposure of molybdenum can cause loss of appetite, tiredness, headaches and muscle soreness. [5] [14]
Molybdenum disulfide (or moly) is an inorganic compound composed of molybdenum and sulfur. Its chemical formula is MoS 2. The compound is classified as a transition metal dichalcogenide. It is a silvery black solid that occurs as the mineral molybdenite, the principal ore for molybdenum. [6] MoS 2 is relatively unreactive.
Alternative veterinary medicine is the use of alternative medicine in the treatment of animals. Types alternative therapies used for veterinary treatments may include, but are not limited to, acupuncture, herbal medicine, homeopathy, ethnomedicine and chiropractic. The term includes many treatments that do not have enough evidence to support ...
Descriptions of human molybdenum deficiency are few. A patient receiving prolonged parenteral nutrition acquired a syndrome described as ‘acquired molybdenum deficiency.’ This syndrome, exacerbated by methionine administration, was characterized by high blood methionine, low blood uric acid , and low urinary uric acid and sulfate ...
Unlike many other cofactors, molybdenum cofactor (Moco) cannot be taken up as a nutrient. The cofactor thus requires de novo biosynthesis.Molybdenum cofactor biosynthesis occurs in four steps: (i) the radical-mediated cyclization of nucleotide, guanosine triphosphate (GTP), to (8S)‑3',8‐cyclo‑7,8‑dihydroguanosine 5'‑triphosphate (3',8‑cH 2 GTP), (ii) the formation of cyclic ...