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Alternatively, autobiographical memory loss may result from subtle changes in the temporal lobe which gives rise to temporal lobe epilepsy and to memory problems. The mechanism and etiology of this phenomenon remain controversial, especially as it is impossible to rule out prior subclinical epileptic activity which could be responsible for a ...
Many different types of memory have been identified in humans, such as declarative memory (including episodic memory and semantic memory), working memory, spatial memory, and procedural memory. [6] Studies done, have found that memory functions, more specifically those associated with the medial temporal lobe are especially vulnerable to age ...
fMRI scans following brief sleep deprivation (24 hours or less) show increases in thalamic activation. Verbal working memory tasks normally cause increases in left temporal lobe activity. After 35 hours come noted decreases in temporal lobe activation and increases in parietal lobe activation. [42] Working memory model.
This is why after a stroke people have a chance of developing cognitive deficits that result in anterograde amnesia, since strokes can involve the temporal lobe in the temporal cortex, and the temporal cortex houses the hippocampus. Anterograde amnesia can be the first clinical sign that Alzheimer's disease is developing within the brain ...
Temporal lobe signs usually involve auditory sensation and memory, and may include: [citation needed] deafness without damage to the structures of the ear, described as cortical deafness; tinnitus, auditory hallucinations; loss of ability to comprehend music or language, described as a sensory aphasia (Wernicke's aphasia)
Lateral temporal lobe seizures arising from the temporal-parietal lobe junction may cause complex visual hallucinations. [2] In comparison to mesial temporal lobe seizures, lateral temporal lobe seizures are briefer duration seizures, occur with earlier loss of awareness, and are more likely become a focal to bilateral tonic-clonic seizure. [2]
Frontal lobe epilepsy; Lacunar syndromes; Migraine variants; Posterior cerebral artery stroke; Syncope and related paroxysmal spells; Temporal lobe epilepsy; If the event lasts less than one hour, transient epileptic amnesia (TEA) might be implicated. [2] [30] If the condition lasts longer than 24 hours, it is not considered TGA by definition.
It is currently unknown why semantic memory is impaired and semantic knowledge deteriorates in SD patients, though the cause may be due to damage to an amodal semantic system. This theory is supported by the atrophy of the anterior temporal lobe, which is believed to contain a component of the semantic system that integrates conceptual information.