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The dentinoenamel junction is thought to be of a scalloped structure which has occurred as an exaptation of the epithelial folding that is undergone during ontogeny. This scalloped exaptation has then provided stress relief during mastication and a reduction in dentin-enamel sliding and has thus, not been selected against, making it an ...
Enamel spindles are "short, linear defects, found at the dentinoenamel junction (DEJ) and extend into the enamel, often being more prevalent at the cusp tips." [1] The DEJ is the interface of the enamel and the underlying dentin.
The lightish boundary between them is the dentinoenamel junction. From this can be seen enamel tufts growing towards the top right. Enamel tufts are hypomineralized ribbon-like structures that run longitudinally to the tooth axis and extend from the dentinoenamel junction (DEJ) one fifth to a third into the enamel. [1]
The dentinoenamel junction is the surface at which the enamel and the dentin of the crown of a tooth are joined. [ 7 ] The existing ectomesenchyme around the outside of the cap of the enamel organ then condenses into the dental sac.
Tooth development begins at week 6 in utero, in the oral epithelium. The process is divided into three stages: Initiation; Morphogenesis and; Histogenesis [2]; At the end of week 7 i.u., localised proliferations of cells in the dental laminae form round and oval swellings known as tooth buds, which will eventually develop into mesenchymal cells and surround the enamel organ.
Amelogenesis is the process of forming tooth enamel, the hard, protective outer layer of teeth. [1] This process begins during tooth development after the initial formation of dentin (dentinogenesis), the layer beneath the enamel. [2]
Thus with the formation of primary dentin, the cell moves pulpally, away from the basement membrane (future dentinoenamel junction) at the interface between the inner enamel epithelium and dental papilla, leaving behind the odontoblastic process within the dentin.
Enamel lamellae are a type of hypomineralized structure in teeth that extend either from the dentinoenamel junction (DEJ) to the surface of the enamel, or vice versa. In essence, they are prominent linear enamel defects, but are of no clinical consequence. [1] These structures contain proteins, proteoglycans, and lipids.