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Autosomal dominant polycystic kidney disease (ADPKD) is the most common of all the inherited cystic kidney diseases [12] [13] [14] with an incidence of 1:500 live births. [12] [14] Studies show that 10% of end-stage kidney disease (ESKD) patients being treated with dialysis in Europe and the U.S. were initially diagnosed and treated for ADPKD ...
A case of fungal infection of the big toe Advanced fungal infection of the big toe. The most common symptom of a fungal nail infection is the nail becoming thickened and discoloured: white, black, yellow or green. As the infection progresses the nail can become brittle, with pieces breaking off or coming away from the toe or finger completely.
Autosomal dominant polycystic kidney disease (ADPKD) is one of the most common, life-threatening inherited human disorders and the most common hereditary kidney disease. [ 1 ] [ 2 ] It is associated with large interfamilial and intrafamilial variability, which can be explained to a large extent by its genetic heterogeneity and modifier genes ...
Often there is a distinction made between conditions of the dorsal skin and plantar skin. Common examples include callus thickened skin, fungal infections of the skin ( athlete's foot ) or nails ( onychomycosis ), viral infection of verrucae , and ingrowing toenails that may cause bacterial nail infections ( paronychia ).
Athlete's foot is the most common fungal disease, with possibly more than 50% of the population affected at some time. [2] [4] Tinea manuum accounts for less than 2% of all superficial fungal infections. [2] Tinea manuum is rare in both hands. [2] Scenarios with one foot and two hands, and one foot and one hand, have been described. [15]
Polycystic kidney disease (ADPKD) is a life threatening hereditary disorder; it is characterized by the development of fluid-filled cyst formation and expansion of the kidney and other organs. [3] It is an autosomal dominant disease, and it is the most common hereditary disorders with a rate of occurrence of approximately 1 in 1000. [4]
The syndrome was discovered in June, 1988 by Dr. G. Exner, [5] a researcher at Orthopädische Universitätsklinik Balgrist in Zurich, Switzerland.Exner officially named the disorder serpentine fibula polycystic kidney syndrome, but the term "Exner syndrome" became more prevalent.
The treatment options for autosomal recessive polycystic kidney disease, given there is no current cure, are: [4] Medications for hypertension; Medications and/or surgery for pain; Antibiotics for infection; Dialysis (if kidney failure is present) Kidney transplantation(in serious cases)