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Stress urinary incontinence is a common problem related to the function of the urethral sphincter. Weak pelvic floor muscles, intrinsic sphincter damage, or damage to the surrounding nerves and tissue can make the urethral sphincter incompetent, and subsequently it will not close fully, leading to stress urinary incontinence.
The internal urethral sphincter is a urethral sphincter muscle which constricts the internal urethral orifice. It is located at the junction of the urethra with the urinary bladder and is continuous with the detrusor muscle , [ 1 ] [ 2 ] but anatomically and functionally fully independent from it. [ 3 ]
The muscle helps maintain continence of urine along with the internal urethral sphincter which is under control of the autonomic nervous system.The external sphincter muscle prevents urine leakage as the muscle is tonically contracted via somatic fibers that originate in Onuf's nucleus and pass through sacral spinal nerves S2-S4 then the pudendal nerve to synapse on the muscle.
The body is calm, the bladder muscles contract, and the internal sphincter, which is located at the opening between the bladder and urethra, opens, letting you pee.
The sphincter urethrae, or urethral sphincter, controlling the exit of urine from the body. At the anus, there are two anal sphincters which control the exit of feces from the body, the internal anal sphincter and external anal sphincter. The inner sphincter is involuntary and the outer is voluntary.
A short (1 or 2 cm) portion passing through the external urethral sphincter. This is the narrowest part of the urethra. It is located in the deep perineal pouch. The bulbourethral glands (Cowper's gland) are found posterior to this region but open in the spongy urethra. Pseudostratified columnar: Spongy urethra (or penile urethra)
Bulbourethral glands are located posterior and lateral to the membranous portion of the urethra at the base of the penis, between the two layers of the fascia of the urogenital diaphragm, in the deep perineal pouch. They are enclosed by transverse fibers of the sphincter urethrae membranaceae muscle.
In normal lower urinary tract function, these two separate muscle structures act in synergistic coordination. But in this neurogenic disorder, the urethral sphincter muscle, instead of relaxing completely during voiding, dyssynergically contracts causing the flow to be interrupted and the bladder pressure to rise. [4]