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Canine subvalvular aortic stenosis (SAS) is an abnormal, congenital heart murmur caused by subaortic stenosis (SAS). This genetic trait is polygenic, and in some cases asymptomatic. An animal with SAS may offspring and show no symptoms of the stenosis until multiple generations which is why it is advised not to breed an animal diagnosed with ...
The congenital heart block is a rare disease that affects around 1 child in every 15,000–20,000 births. [3] However, its high mortality (which can be as high as 85% in some severe cases) makes the early diagnosis and intervention very important. [ 1 ]
It is a congenital heart disease in dogs. There usually are no signs in dogs except for a heart murmur. However, a large defect can result in heart failure or in pulmonary hypertension leading to a right-to-left shunt. [49] Atrial septal defect* is a hole in the division between the heart atria (upper chambers of the heart). It is an uncommon ...
Eisenmenger syndrome or Eisenmenger's syndrome is defined as the process in which a long-standing left-to-right cardiac shunt caused by a congenital heart defect (typically by a ventricular septal defect, atrial septal defect, or less commonly, patent ductus arteriosus) causes pulmonary hypertension [1] [2] and eventual reversal of the shunt into a cyanotic right-to-left shunt.
The cause of congenital third-degree heart block in many patients is unknown. Studies suggest that the prevalence of congenital third-degree heart block is between 1 in 15,000 and 1 in 22,000 live births. [citation needed] Hyperkalemia in those with previous cardiac disease [8] and Lyme disease can also result in third-degree heart block. [9]
Ebstein's anomaly is a congenital heart defect in which the septal and posterior leaflets of the tricuspid valve are displaced downwards towards the apex of the right ventricle of the heart. [1] EA has great anatomical heterogeneity that generates a wide spectrum of clinical features at presentation and is complicated by the fact that the ...
Due to non-compaction cardiomyopathy being a relatively new disease, its impact on human life expectancy is not very well understood. In a 2005 study [ 3 ] that documented the long-term follow-up of 34 patients with NCC, 35% had died at the age of 42 +/- 40 months, with a further 12% having to undergo a heart transplant due to heart failure.
Out of all the babies born with congenital heart defects, about 25 % have cyanosis as a result. Tetralogy of Fallot is the most common cyanotic cardiac heart defect. [37] Methemoglobinemia is considered to be rare, with acquired methemoglobinemia encountered more than the congenital form. [38]