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Memory consolidation was first referred to in the writings of the renowned Roman teacher of rhetoric Quintillian.He noted the "curious fact... that the interval of a single night will greatly increase the strength of the memory," and presented the possibility that "... the power of recollection .. undergoes a process of ripening and maturing during the time which intervenes."
Other species similarly tested with some measure of success include goldfish (discriminating between 2 vs. 3, and 10 vs. 15), [79] guppy (3 vs. 4, and 4 vs. 5), [80] [81] and zebrafish (2 vs. 3, 3 vs. 4, and 4 vs. 5, but not 5 vs. 6, nor 6 vs. 7). [82] Many studies have shown that when given a choice, shoaling fish prefer to join the larger of ...
This is in opposition to the classical consolidation hypothesis which regards consolidation as a one-time event, following the first encoding of a memory. A memory item in this hypothesis, upon reactivation, destabilizes for a brief period and thereafter invokes the neuronal processes requisite for stabilization. [5]
This slow process, referred to as consolidation, allows emotions to influence the way the memory is stored. [7] The amygdala is involved in memory consolidation, which is the process of transferring information that is currently in working memory into ones long-term memory. This process is also known as memory modulation. [7]
Moreover the transgenic mice performed normally on memory tasks 30 minutes after training, but were considerably impaired 24 hours after training. This led to the conclusion that calcineurin-overexpressing mice have a deficit in long-term memory consolidation, which reflects their deficit in late phase LTP.
A so-called "doomsday fish" has washed up on a Southern California beach — typically an extremely rare occurrence, but this is the second time this year it has happened. The rare oarfish found ...
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LTP describes cellular level consolidation, which is the consolidation of a memory that occurs between individual neurons. Initially, cellular consolidation, or LTP, begins in the hippocampus; there, protein synthesis inhibitors, tetrodotoxin, lidocaine, lesions and other factors can interfere with hippocampal activity and cause memory deficits ...