Search results
Results from the WOW.Com Content Network
[2] [9] This distal attachment is the stronger of the two and makes up the floor of the pes anserine bursa. The proximal tibial attachment of the sMCL is the primary stabilizer to valgus force on the knee, whereas the distal tibial attachment is the primary stabilizer of external rotation at 30° of knee flexion. [3] [9]
Diagram showing named veins. Perforator veins exist along the length of the lower limb, in greater number in the leg (anatomical ref to below knee) than in the thigh. Some veins are named after the physician who first described them: Dodd's perforator at the inferior 1/3 of the thigh; Boyd's perforator at the knee level
The permeability and compressibility of the superficial vein system (SVS), the presence or absence of superficial insufficiency, and in which veins or vein segments; Which perforator veins are continent or insufficient; The presence or absence of shunts; Mapping the insufficient veins, flux direction, shunts, and perforators. [27] [nb 4]
The gastrocnemius muscle is prone to spasms, which are painful, involuntary contractions of the muscle that may last several minutes. [5] A severe ankle dorsiflexion force may result in a Medial Gastrocnemius Strain (MGS) injury of the muscle, commonly referred to as a "torn" or "strained" calf muscle, which is acutely painful and disabling. [6]
Risk factors include bone fractures, joint dislocations, alcoholism, and the use of high-dose steroids. [1] The condition may also occur without any clear reason. [1] The most commonly affected bone is the femur (thigh bone). [1] Other relatively common sites include the upper arm bone, knee, shoulder, and ankle. [1]
The fibular collateral ligament (FCL) connects the femur to the fibula. It attaches on the femur just proximal and posterior to the femoral lateral epicondyle and extends approximately 70 mm down the knee to attach to the fibular head. [10] [11] From 0° to 30° of knee flexion, the FCL is the main structure preventing varus opening of the knee ...
Chronic venous insufficiency (CVI) is a medical condition characterized by blood pooling in the veins, leading to increased pressure and strain on the vein walls. [1] The most common cause of CVI is superficial venous reflux, which often results in the formation of varicose veins, a treatable condition. [2]
This valvular incompetence combined with persistent venous obstruction from thrombus increases the pressure in veins and capillaries. Venous hypertension induces a rupture of small superficial veins, subcutaneous hemorrhage [7] and an increase of tissue permeability. That is manifested by pain, swelling, discoloration, and even ulceration.