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The dental follicle, also known as dental sac, is made up of mesenchymal cells and fibres surrounding the enamel organ and dental papilla of a developing tooth. [1] It is a vascular fibrous sac [2] containing the developing tooth and its odontogenic organ. The dental follicle (DF) differentiates into the periodontal ligament.
B: dental papilla C: dental follicle. The tooth germ is an aggregation of cells that eventually forms a tooth. [2] These cells are derived from the ectoderm of the first pharyngeal arch and the ectomesenchyme of the neural crest. [1] [3] [4] The tooth germ is organized into three parts: the enamel organ, the dental papilla and the dental sac or ...
The dental follicle is a fibrous sac that surrounds the odontogenic organ and developing tooth. [11] The plexus is a system of connections of blood vessels, nerves, or lymphatic vessels . The plexus of Raschkow is a network of nerves immediately beneath the odontoblast layer of the dentine, first described by J. Raschkow in 1835.
The dental papilla: below the enamel organ; The tooth sac: ectomesenchymal condensation of the area surrounding the tooth bud and dental papilla. Both the dental papilla and the tooth sac are not structurally defined in the bud stage, and will become more defined in subsequent stages (Cap and Bell stages).
When impacted teeth are retained completely within the alveolar process, the associated follicular sac is also retained along with it. Though in most persons the dental follicle maintains its original size sometimes it may undergo cystic degeneration and become a dentigerous cyst or a keratocyst.
HERS is a collar of epithelial cells derived from the apical prolongation of the enamel organ. Once the root sheath disintegrates, the newly formed surface of root dentin comes into contact with the undifferentiated cells of the dental sac (dental follicle). This then stimulates the activation of cementoblasts to begin cementogenesis. The ...
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Another possible example of this sensitivity (stress response pathway activation) may be the development of dental fluorosis after childhood exposure (between the ages of 2 and 8 years old) to excess consumption of fluoride, an elemental agent used to increase enamel hardness and as a result, prevent dental caries.