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The contralateral (opposite) leg then bears all of the weight, which reduces blood flow to the hoof and strains the attachments of the laminae, leading to laminitis. Although support-limb laminitis is a risk for any horse that is not weight-bearing lame, occurring in roughly 16% of cases, it is uncommon in foals and yearlings. [135]
A horse can live with laminitis for many years, and although a single episode of laminitis predisposes to further episodes, with good management and prompt treatment it is by no means the catastrophe sometimes supposed: most horses suffering an acute episode without pedal bone displacement make a complete functional recovery. Some ...
Appropriate treatment for lameness depends on the condition diagnosed, but at a minimum it usually includes rest or decreased activity and anti-inflammatory medications. Other treatment options, such as corrective shoeing, joint injections, and regenerative therapies, are pursued based on the cause of lameness and the financial limits of the owner.
The prognosis for a horse with navicular syndrome is guarded. Many times the horse does not return to its former level of competition. Others are retired. Eventually all horses with the syndrome will need to lessen the strenuousness of their work, but with proper management, a horse with navicular syndrome can remain useful for some time.
This pony has a body condition suggestive of EMS. Equine metabolic syndrome (EMS) is an endocrinopathy affecting horses and ponies.It is of primary concern due to its link to obesity, insulin dysregulation, and subsequent laminitis.
Plants can cause reactions ranging from laminitis (found in horses bedded on shavings from black walnut trees), anemia, kidney disease and kidney failure (from eating the wilted leaves of red maples), to cyanide poisoning (from the ingestion of plant matter from members of the genus Prunus) and other symptoms.
However, any horse that can store excess amounts of glycogen, usually genetic, can develop this form of ER. [1] Recurrent Exertional Rhabdomyolysis (RER) is commonly found in breeds that are high strung such as Arabians and thoroughbreds. However, any horse can develop this type of ER if it displays abnormal muscle contractions. [1]
A horse with PPID, which has an overactive pars intermedia not regulated by glucocorticoid levels, does not suppress ACTH production and, therefore, cortisol levels remain high. False negatives can occur in early disease. [1] Additionally, dexamethasone administration may increase the risk of laminitis in horses already prone to the disease. [8]