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A silent stroke (or asymptomatic cerebral infarction) is a stroke that does not have any outward symptoms associated with stroke, and the patient is typically unaware they have suffered a stroke. Despite not causing identifiable symptoms, a silent stroke still causes damage to the brain and places the patient at increased risk for both ...
Cerebral infarction, also known as an ischemic stroke, is the pathologic process that results in an area of necrotic tissue in the brain (cerebral infarct). [1] In mid to high income countries, a stroke is the main reason for disability among people and the 2nd cause of death. [ 2 ]
In a select population with symptoms secondary to a high outflow tract gradient, alcohol septal ablation can reduce the symptoms of HCM. In addition, older individuals and those with other medical problems, for whom surgical myectomy would pose increased procedural risk, would likely benefit from the less-invasive septal ablation procedure.
Myocardial infarction complications may occur immediately following a myocardial infarction (heart attack) (in the acute phase), or may need time to develop (a chronic problem). After an infarction, an obvious complication is a second infarction, which may occur in the domain of another atherosclerotic coronary artery, or in the same zone if ...
Myocardial ruptures can be classified as one of three types. [citation needed] Type I myocardial rupture is an abrupt, slit-like tear that generally occurs within 24 hours of an acute myocardial infarction. Type II is an erosion of the infarcted myocardium, which is suggestive of a slow tear of the dead myocardium.
A VSD can cause a left-to-right shunt of blood flow in the heart and is one of the most common of the congenital heart defects. This type of shunt is an acyanotic disorder that can result in ventricular hypertrophy. [4] The alignment of interventricular septum and interatrial septum is disturbed in various congenital heart diseases. [5]
A myocardial infarction in the setting of a paradoxical embolism are caused by the emboli disrupting blood flow in a coronary artery. Physical findings that should be evaluated include a comprehensive neurological examination for evaluation of stroke symptoms such as weakness, gait changes, slurred speech, and facial droop.
The dead tissue is surrounded by a zone of potentially reversible ischemia that progresses to become a full-thickness transmural infarct. [76] [79] The initial "wave" of infarction can take place over 3–4 hours. [73] [76] These changes are seen on gross pathology and cannot be predicted by the presence or absence of Q waves on an ECG. [78]