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The remaining 50% are due to non-biliary causes. This is because upper abdominal pain and gallstones are both common but are not always related. Non-biliary causes of PCS may be caused by a functional gastrointestinal disorder, such as functional dyspepsia. [6] Chronic diarrhea in postcholecystectomy syndrome is a type of bile acid diarrhea ...
Osmotic diarrhea, distension of the small bowel leading to crampy abdominal pain, and reduced blood volume can result. Late dumping syndrome occurs 2 to 3 hours after a meal. It results from excessive movement of sugar into the intestine, which raises the body's blood glucose level and causes the pancreas to increase its release of the hormone ...
In patients with persistent afferent loop syndrome, steatorrhea and diarrhea may exacerbate intestinal stasis. Iron deficiency anemia and/or vitamin B12 deficiency can arise from the deconjugation of bile salts by bacteria. In order to avoid postprandial pain, patients frequently quit eating, which can result in significant weight loss. [2]
The first step in diagnosis is to determine the etiology of abdominal distension. After making a differential diagnosis of abdominal distension, it is important to take a careful medical history. [9] Here are the most common causes of abdominal distension classified as an underlying cause and as a secondary disease. As an underlying disease cause:
Early satiety is the sensation of stomach fullness that occurs shortly after beginning to eat and is out of proportion to the meal. [10] Bloating is a highly subjective feeling of increased abdominal pressure. Bloating without eating should be distinguished from postprandial fullness. It is sometimes, but not always, associated with food ...
The gastrocolic reflex or gastrocolic response is a physiological reflex that controls the motility, or peristalsis, of the gastrointestinal tract following a meal. It involves an increase in motility of the colon consisting primarily of giant migrating contractions, in response to stretch in the stomach following ingestion and byproducts of digestion entering the small intestine. [1]
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The chronic form typically presents more gradually with abdominal pain after eating, unintentional weight loss, vomiting, and fear of eating. [ 1 ] [ 2 ] Risk factors for acute intestinal ischemia include atrial fibrillation , heart failure , chronic kidney failure , being prone to forming blood clots , and previous myocardial infarction . [ 2 ]