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Kidney function gradually decreases as someone ages. The elderly are also likely to be underweight. In addition, these older people tend to be dehydrated and be taking other medications. These factors increase the likelihood of developing side effects of digoxin and digoxin toxicity. Often lowering the dose is considered by the prescriber. [6]
If the vomiting reflex continues for an extended period with no appreciable vomitus, the condition is known as non-productive emesis or "dry heaves", which can be painful and debilitating. [citation needed] Color of vomit [16] Bright red in the vomit suggests bleeding from the esophagus
A disulfiram-like drug is a drug that causes an adverse reaction to alcohol leading to nausea, vomiting, flushing, dizziness, throbbing headache, chest and abdominal discomfort, and general hangover-like symptoms among others.
They may include vomiting, loss of appetite, confusion, blurred vision, changes in color perception, and decreased energy. [1] Potential complications include an irregular heartbeat, which can be either too fast or too slow. [1] Toxicity may occur over a short period of time following an overdose or gradually during long-term treatment. [1]
If the wikilink of that drug name redirects to another drug name (eg trade name to generic) then mention it on the list. If a trade name should redirect to a generic name of a drug page that hasn't been created yet, include the redirect mention in the list but don't create a page that redirects to a non-existant page.
These drugs block one or more of the nerve signals that cause nausea and vomiting. During the first 24 hours after chemotherapy, the most effective approach appears to be blocking the 5-HT 3 nerve signal. [10] Approved 5-HT 3 inhibitors include dolasetron (Anzemet), granisetron (Kytril, Sancuso), and ondansetron (Zofran). Their antiemetic ...
Cantharidin is known to cause hematemesis (vomiting of blood) and gross hematuria (blood visible in urine). It can also cause dysuria (pain while urinating). Priapism.
Drug-induced pigmentation of the skin may occur as a consequence of drug administration, and the mechanism may be postinflammatory hyperpigmentation in some cases, but frequently is related to actual deposition of the offending drug in the skin. [2]: 125–6 The incidence of this change varies, and depends on the type of medication involved.