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The delay in eruption has been suggested as the reason for the apparent difference in decay among the youngest children. Fluoride ingestion during tooth development can lead to a permanent condition known as fluorosis with varying levels of severity, the result of fluoride's interference with the normal osteoblast development. [56] [57] [58 ...
Anodontia is the total lack of tooth development. Hyperdontia is the presence of a higher-than-normal number of teeth. Hypodontia is the lack of development of one or more teeth. Oligodontia may be used to describe the absence of 6 or more teeth.
Tooth development is commonly divided into the following stages: the bud stage, the cap, the bell, and finally maturation. The staging of tooth development is an attempt to categorize changes that take place along a continuum; frequently it is difficult to decide what stage should be assigned to a particular developing tooth. [5]
Tooth development begins at week 6 in utero, in the oral epithelium. The process is divided into three stages: Initiation; Morphogenesis and; Histogenesis [2]; At the end of week 7 i.u., localised proliferations of cells in the dental laminae form round and oval swellings known as tooth buds, which will eventually develop into mesenchymal cells and surround the enamel organ.
Schematic of maxillary lateral incisors in the human mouth for both permanent and primary teeth. Maxillary lateral incisor agenesis (MLIA) is lack of development of one or both of the maxillary lateral incisor teeth. In normal human dentition, this would be the second tooth on either side from the center of the top row of teeth. The condition ...
Bottom teeth of a seven-year-old, showing primary teeth (left), a lost primary tooth (middle), and a fully erupted permanent tooth (right) Although tooth eruption occurs at different times for different people, a general eruption timeline exists. The tooth buds of baby teeth start to develop around 6 weeks of pregnancy.
Radiographically, affected teeth have short and narrow roots, and obliterated pulps due to dentine hypertrophy before or shortly after tooth eruption. [2] The severity of these features is variable, with some teeth presenting with total obliteration of the pulp, while other teeth appear to have normal, healthy dentine. [2]
A delay in tooth development may also serve as an indication, whereby the absence of an adult successor slows down the normal resorption of the roots of the baby teeth, which is the progressive loss of parts of the tooth.
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