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Bladder sphincter dyssynergia (also known as detrusor sphincter dyssynergia (DSD) (the ICS standard terminology agreed 1998) [1] and neurogenic detrusor overactivity (NDO)) is a consequence of a neurological pathology such as spinal injury [2] or multiple sclerosis [3] which disrupts central nervous system regulation of the micturition (urination) reflex resulting in dyscoordination of the ...
Bladder sphincter dyssynergia also known as detrusor sphincter dyssynergia is the decrease of detrusor (wall muscle of the bladder) pressure which causes unwanted urination. This is very common in spinal cord injuries and multiple sclerosis patients. There is a malfunction between the central nervous system, urinary sphincters, and detrusor ...
Pseudodyssynergia (or detrusor sphincter pseudodyssynergia) is an urological condition involving contraction of the male or female external sphincter during voiding. [1] Coordination between the sphincter and detrusor is thus lost. [2]
[6] [11] Urethral pressure monitoring is another less-invasive approach to assessing detrusor sphincter dyssynergia. [11] These studies can be repeated at regular intervals, especially if symptoms worsen or to measure response to therapies. [9] Evaluation of kidney function through blood tests such as serum creatinine should be obtained. [6]
Without diagnostic evaluation, the cause of underactive bladder is unclear, as there are multiple possible causes. UAB symptoms can accurately reflect impaired bladder emptying due either to DU or obstruction (normal or large storage volumes, elevated post-void residual volume), or can result from a sense of incomplete emptying of a hypersensitive bladder (small storage volumes, normal or ...
Cerebellar ataxia is a form of ataxia originating in the cerebellum. [1] Non-progressive congenital ataxia (NPCA) is a classical presentation of cerebral ataxias.. Cerebellar ataxia can occur as a result of many diseases and may present with symptoms of an inability to coordinate balance, gait, extremity and eye movements. [2]
Onset of symptoms usually occurs in early adulthood and is characterized by intention tremor, progressive ataxia, convulsions, and myoclonic epileptic jerks. [1] Tremors usually affect one extremity, primarily the upper limb, and eventually involve the entire voluntary motor system. [ 1 ]
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