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  2. Biological response modifier - Wikipedia

    en.wikipedia.org/wiki/Biological_response_modifier

    Biological response modifiers (BRMs) are substances that modify immune responses. They can be endogenous (produced naturally within the body) or exogenous (as pharmaceutical drugs ), and they can either enhance an immune response or suppress it .

  3. Immunosuppressive drug - Wikipedia

    en.wikipedia.org/wiki/Immunosuppressive_drug

    Interleukin-2 is an important immune system regulator necessary for the clone expansion and survival of activated lymphocytes T. Its effects are mediated by the trimer cell surface receptor IL-2a, consisting of the α, β, and γ chains. The IL-2a (CD25, T-cell activation antigen, TAC) is expressed only by the already-activated T lymphocytes.

  4. Type 2 inflammation - Wikipedia

    en.wikipedia.org/wiki/Type_2_inflammation

    Type 2 inflammation is a pattern of immune response. Its physiological function is to defend the body against helminths, but a dysregulation of the type 2 inflammatory response has been implicated in the pathophysiology of several diseases. [1] [2]

  5. Immunogenicity - Wikipedia

    en.wikipedia.org/wiki/Immunogenicity

    However, strictly speaking, immunogenicity refers to the ability of an antigen to induce an adaptive immune response. Thus an antigen might bind specifically to a T or B cell receptor, but not induce an adaptive immune response. If the antigen does induce a response, it is an 'immunogenic antigen', which is referred to as an immunogen.

  6. Immune response - Wikipedia

    en.wikipedia.org/wiki/Immune_response

    An immune response is a physiological reaction which occurs within an organism in the context of inflammation for the purpose of defending against exogenous factors. These include a wide variety of different toxins, viruses, intra- and extracellular bacteria, protozoa, helminths, and fungi which could cause serious problems to the health of the host organism if not cleared from the body.

  7. Receptor theory - Wikipedia

    en.wikipedia.org/wiki/Receptor_theory

    However, he later modified the theory in order to explain immune reactions and the selectivity of the immune response. [6] Thinking that selectivity was derived from interaction with the tissues themselves, Ehrlich envisaged molecules extending from cells that the body could use to distinguish and mount an immune response to foreign objects.

  8. Damage-associated molecular pattern - Wikipedia

    en.wikipedia.org/wiki/Damage-associated...

    Once a DAMP is released from the cell, it promotes a noninfectious inflammatory response by binding to a pattern recognition receptor (PRR). [4] Inflammation is a key aspect of the innate immune response; it is used to help mitigate future damage to the organism by removing harmful invaders from the affected area and start the healing process. [5]

  9. Antigen-antibody interaction - Wikipedia

    en.wikipedia.org/wiki/Antigen-antibody_interaction

    Acquired immunity depends upon the interaction between antigens and a group of proteins called antibodies produced by B cells of the blood. There are many antibodies and each is specific for a particular type of antigen. Thus immune response in acquired immunity is due to the precise binding of antigens to antibody.