Search results
Results from the WOW.Com Content Network
HHV-6A and HHV-6B were recognized by the International Committee on Taxonomy of Viruses (ICTV) as distinct species in 2012. Human roseoloviruses include HHV-6A, HHV-6B and HHV-7. [1] Herpesvirus was established as a genus in 1971 in the first report of the ICTV. This genus consisted of 23 viruses among 4 groups. [22]
Human betaherpesvirus 6A (HHV-6A) is a species of virus in the genus Roseolovirus, subfamily Betaherpesvirinae, family Herpesviridae, and order Herpesvirales. [ 1 ] HHV-6A and infertility
[8] [9] [10] Other human herpesviruses are human herpesvirus 6A and 6B (HHV-6A and HHV-6B) and human herpesvirus 7 (HHV-7), which are the etiological agents for Roseola, and HHV-8 (also known as KSHV) which is responsible for causing Kaposi's sarcoma-associated herpesvirus. [7] HHV here stands for "Human Herpesvirus".
[5] Human herpesvirus 6A (HHV-6A) has been described as more neurovirulent, [6] and as such is more frequently found in patients with neuroinflammatory diseases such as multiple sclerosis. [7] Both human herpesvirus 6B (HHV-6B) and human herpesvirus 7 (HHV-7), as well as other viruses, can cause a skin condition in infants known as exanthema ...
For premium support please call: 800-290-4726 more ways to reach us
Human herpesvirus 6 (HHV-6) is a set of two closely related herpes viruses known as HHV-6A and HHV-6B that infect nearly all human beings, typically before the age of two. The acquisition of HHV-6 in infancy is often symptomatic, resulting in childhood fever, diarrhea, and exanthem subitum rash (commonly known as roseola).
The incubation period of the virus can be three to six days, according to the CDC, and the groups at highest risk include younger children and adults 65 or older.
HHV-6 has been further classified into HHV-6A and HHV-6B, two distinct viruses which share 88% of the same DNA makeup, with HHV-6B the most common cause of roseola. [4] [13] After infection, these viruses enter a latent phase. Roseola caused by HHV-7 has been linked to the ability of HHV-7 infection to reactivate latent HHV-6. [6]