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AV-nodal reentrant tachycardia (AVNRT) is a type of abnormal fast heart rhythm. It is a type of supraventricular tachycardia (SVT), meaning that it originates from a location within the heart above the bundle of His. AV nodal reentrant tachycardia is the most common regular supraventricular tachycardia.
AVRT stands for atrioventricular reentrant tachycardia, which is a fast heart rate that results from an accessory electrical pathway in the heart. AVNRT stands for atrioventricular nodal reentrant tachycardia, and results from a reentrant circuit that's in or near the AV node.
Another type of reentrant circuit, though, is atrioventricular nodal reentrant tachycardia, or AVNRT. AVNRT, just like AVRT, is a type of supraventricular tachycardia, but with AVNRT it’s in or near the AV node, which just like before contracts the ventricle and the atria every time it goes around.
Atrial flutter is also not necessarily a tachycardia by definition unless the AV node permits a ventricular response greater than 100 beats per minute. AV nodal reentrant tachycardia (AVNRT) involves a reentry circuit forming next to, or within, the AV node. The circuit most often involves two tiny pathways one faster than the other.
In the absence of shock, inhibition at the AV node is attempted. This is achieved first by a trial of specific physical maneuvers such as holding a breath in or bearing down. If these maneuvers fail, using intravenous adenosine [ 4 ] causes complete electrical blockade at the AV node and interrupts the reentrant electrical circuit.
AV nodal reentrant tachycardia (AVNRT) is the most common reentrant tachycardia. It is a regular narrow complex tachycardia that usually responds well to the Valsalva maneuver or the drug adenosine. However, unstable patients sometimes require synchronized cardioversion.
The majority of time symptomatic WPW fits the definition of AVRT (Supraventricular tachycardia) however AVNRT (dual AV nodal physiology) exist in ~10% of patients with WPW syndrome creating the possibility of spontaneous atrial fibrillation degenerating into ventricular fibrillation (VF). The fact that WPW patients are young and do not have ...
The underlying anatomical causes of PSVT, specifically atrioventricular nodal reentry, involve the presence of two functionally distinct conduction pathways within the AV node and the formation of a reentrant circuit that sustains the tachycardia. [8] These pathways within the AV node are known as the fast pathway and the slow pathway. [9]