Search results
Results from the WOW.Com Content Network
Crystalluria refers to crystals found in the urine when performing a urine test. Crystalluria is considered often as a benign condition and as one of the side effects of sulfonamides and penicillins. The main reason for the identification of urinary crystals is to detect the presence of the relatively few abnormal types that may represent a ...
The presence of crystals in the urine has conventionally been associated with the formation of kidney stones, and crystalluria is more common in people with kidney stones than those without. However, crystalluria occurs in up to 20% of the normal population, so it is not a reliable diagnostic marker. [126]
Antibacterials like dapsone (increases plasma levels of both drugs), methenamine (increased risk of crystalluria) and rifampicin (as it may lead to an increased plasma level of rifampicin and lower plasma levels of trimethoprim) Anticoagulants like warfarin and acenocoumarol — anticoagulant effects of either drug is potentiated by this ...
The massive crystalluria that sometimes occurs sets its symptom profile apart from that of phenobarbital. [51] [53] [54] [55] The crystals are white, [52] [54] needle-like, [53] shimmering, hexagonal plates consisting mainly of primidone. [52] [54] In the Netherlands alone, 34 cases of suspected primidone poisoning occurred between 1978 and 1982.
Although struvite was briefly mentioned in Hooke's Micrographia, [5] it was first described in detail in 1845 by the German chemist Georg Ludwig Ulex [] (1811–1883), who found crystals of struvite in what he surmised had once been a medieval midden in Hamburg, Germany; he named the new mineral after the geographer and geologist Heinrich Christian Gottfried von Struve [] (1772–1851) of Hamburg.
Bacteriuria is the presence of bacteria in urine. [1] Bacteriuria accompanied by symptoms is a urinary tract infection while that without is known as asymptomatic bacteriuria.
Crystalluria (formation of crystals and excretion in the urine) and acute kidney injury have also been seen. Adverse central nervous system effects are frequent and include confusion, hallucinations, psychosis, ataxia, hearing loss, headache, paresthesia, parkinsonism, peripheral neuropathy, vertigo and sedation.
Hyperoxaluria can be primary (as a result of a genetic defect) or secondary to another disease process. [citation needed]Type I primary hyperoxaluria (PH1) is associated mutations in the gene encoding AGXT, a key enzyme involved in oxalate metabolism.