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A lichenoid eruption is a skin disease characterized by damage and infiltration between the epidermis and dermis. [1] Examples include lichen planus, lichen sclerosus and lichen nitidus. It can also be associated with abrasion or drug use. [2]
Image at right shows influx of inflammatory cells including eosinophils and neutrophils in the dermis (solid arrow) and blister cavity (dashed arrows), and deposition of fibrin (asterisks). [15] However, the diagnosis of bullous pemphigoid consist of at least 2 positive results out of 3 criteria: [19] Pruritus and/or predominant cutaneous blisters
Lymphocytic esophagitis is a rare and poorly understood medical disorder involving inflammation in the esophagus. The disease is named from the primary inflammatory process, wherein lymphocytes are seen within the esophageal mucosa. Symptoms of the condition include difficulty swallowing, heartburn and food bolus obstruction. The condition was ...
Lichen sclerosus (LS) is a chronic, inflammatory skin disease, of unknown cause, which can affect any body part of any person, but has a strong preference for the genitals (penis, vulva), and is also known as balanitis xerotica obliterans when it affects the penis. Lichen sclerosus is not contagious.
Mild inflammatory cell infiltrate along the dermoepidermal junction (black arrow in image) Vacuolization within the basal keratinocytes (white arrow in image) Often necrotic, predominantly basal, individual keratinocytes, manifesting as colloid or Civatte bodies; Acute graft-versus-host-disease
Histopathological image of dyshidrotic dermatitis, showing focal spongiotic change in the epidermis. Spongiosis is mainly intercellular [1] edema (abnormal accumulation of fluid) in the epidermis, [2] and is characteristic of eczematous dermatitis, manifested clinically by intraepidermal vesicles (fluid-containing spaces), "juicy" papules, and/or lichenification. [3]
Biopsies of the lesions show hypertrophied stratified squamous mucosa with glycogen deposition in the mucosa. [1] Clinically, mild glycogenic acanthosis is a normal finding, and does not progress to esophageal cancer or to stricture. [4] It is not related to leukoplakia, and is not dysplastic or premalignant.
Diagnosis often requires a combination of clinical and pathological studies. Diagnosis is sometimes difficult because the early phases of the disease often resemble inflammatory dermatoses (such as eczema , psoriasis , lichenoid dermatoses including lichen planus , vitiligo , and chronic cutaneous lupus erythematosus), as well as other ...