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Changes to chromosome 5 include an extra segment of the short (p) or long (q) arm of the chromosome in each cell (partial trisomy 5p or 5q), a missing segment of the long arm of the chromosome in each cell (partial monosomy 5q), and a circular structure called ring chromosome 5. A ring chromosome occurs when both ends of a broken chromosome are ...
The chromosome is ~193 megabases in length. In a 2012 paper, 775 protein-encoding genes were identified on this chromosome. [4] 211 (27.9%) of these coding sequences did not have any experimental evidence at the protein level, in 2012. 271 appear to be membrane proteins. 54 have been classified as cancer-associated proteins.
The Indian egg-eating snake or Indian egg-eater (Elachistodon westermanni) is a rare species of egg-eating snake in the family Colubridae. The species is endemic to the Indian subcontinent. It is also called Westermann's snake, reflecting its scientific name. The snake belongs to the monotypic genus Elachistodon.
Klinefelter syndrome (KS), also known as 47,XXY, is a chromosome anomaly where a male has an extra X chromosome. [10] These complications commonly include infertility and small, poorly functioning testicles (if present).
Chromosome 21 is one of the 23 pairs of chromosomes in humans.Chromosome 21 is both the smallest human autosome and chromosome, [4] with 46.7 million base pairs (the building material of DNA) representing about 1.5 percent of the total DNA in cells.
Deletion on a chromosome. In genetics, a deletion (also called gene deletion, deficiency, or deletion mutation) (sign: Δ) is a mutation (a genetic aberration) in which a part of a chromosome or a sequence of DNA is left out during DNA replication.
After cooking, it can be considered a protein gel (depending on the length of time it was cooked). Heating high-protein food such as balut can cause the chemical changes to take place and fully or partially denature proteins, causing the surface to become thick and causing an irreversible gel protein to form. [11]
Women with trisomy X who were diagnosed prenatally have better outcomes as a group than those diagnosed postnatally, and 46,XX/47,XXX mosaics better than those with full trisomy X. [3] Some of the improved outcome in prenatal diagnosis appears to be a function of higher socioeconomic status amongst parents.