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The Singapore Medical Association (abbreviated SMA) is a professional association representing the interests of medical professionals in Singapore. It was established on September 15, 1959, replacing the Malaya Branch of the British Medical Association. [2] As of 2020, it had over 8,200 members. [3]
Singapore Medical Association This page was last edited on 9 February 2020, at 01:25 (UTC). Text is available under the Creative Commons Attribution-ShareAlike ...
In 2000, Singapore was ranked 6th in the World Health Organization's ranking of the world's health systems. [1] Bloomberg ranked Singapore's healthcare system the most efficient in the world in 2014. [2] The Economist Intelligence Unit placed Singapore 2nd out of 166 countries for health-care outcomes. [3] Bloomberg Global Health Index of 163 ...
The Singapore Medical Journal is a monthly peer-reviewed general medical journal. It was established in 1960 and is published by Medknow Publications on behalf of the Singapore Medical Association. The editor-in-chief is Poh Kian Keong. According to the Journal Citation Reports, the journal has a 2021 impact factor of 3.331. [1]
Medical research institutes in Singapore (3 P) Pages in category "Medical and health organisations based in Singapore" The following 32 pages are in this category, out of 32 total.
[58] [59] In order to keep the sport complex in frequent use, several plans have been raised by the government to encourage more sporting events into the city, including the purchase of an association football club, Persijatim Solo F.C in 2004 which then renamed to Sriwijaya F.C. [60] Palembang also planned to build a race track inside the ...
11q13.3: Autosomal recessive: Affects mainly infant boys, similar to SMA type 1 but with diaphragmatic paralysis Distal spinal muscular atrophy type 2 (DSMA2) Distal hereditary motor neuronopathy – Jerash type (DHMN-J) 605726: SIGMAR1: 19p13.3: Autosomal recessive: Slowly progressive Distal spinal muscular atrophy type 3 (DSMA3)
The R-Smads consist of Smad1, Smad2, Smad3, Smad5 and Smad8/9, [3] and are involved in direct signaling from the TGF-B receptor. [4] Smad4 is the only known human Co-Smad, and has the role of partnering with R-Smads to recruit co-regulators to the complex. [5] Finally, Smad6 and Smad7 are I-Smads that work to suppress the activity of R-Smads.