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Pyloric stenosis is a narrowing of the opening from the stomach to the first part of the small intestine (the pylorus). [1] Symptoms include projectile vomiting without the presence of bile. [1] This most often occurs after the baby is fed. [1] The typical age that symptoms become obvious is two to twelve weeks old. [1]
In children, congenital pyloric stenosis / congenital hypertrophic pyloric stenosis may be a cause. A pancreatic pseudocyst can cause gastric compression. Pyloric mucosal diaphragm could be a rare cause. Malignant Tumours of the stomach, including adenocarcinoma (and its linitis plastica variant), lymphoma, and gastrointestinal stromal tumours
The symptoms of early and late dumping syndrome are different and vary from person to person. Early dumping syndrome symptoms may include: [1] nausea; vomiting; abdominal pain and cramping; diarrhea; feeling uncomfortably full or bloated after a meal; sweating; weakness; dizziness; flushing, or blushing of the face or skin; rapid or irregular ...
[5] [1] Hypertrophic Pyloric stenosis is a gastrointestinal tract defect, most commonly seen in young children, typically in the first few months of life, caused by enlargement of the tissue in the pyloric muscle. [5] [4] [1] This causes the contents of the stomach to be unable to empty leading to pain after eating, electrolyte abnormalities ...
The pyloric tit sign is a radiological finding observed during barium studies in cases of hypertrophic pyloric stenosis. [1] It appears as an outpouching on the lesser curvature of the stomach, just proximal to the impression created by the hypertrophied pyloric muscle.
The most common symptoms of a duodenal ulcer are waking at night with upper abdominal pain, and upper abdominal pain that improves with eating. [1] With a gastric ulcer, the pain may worsen with eating. [7] The pain is often described as a burning or dull ache. [1] Other symptoms include belching, vomiting, weight loss, or poor appetite. [1]
The definitive treatment for duodenal atresia is surgery (duodenoduodenostomy or duodenojejunostomy), which may be performed openly or laparoscopically. [14] The surgery is required but not immediately urgent - a 24 to 48-hour delay is permissible to facilitate transport, further evaluation and fluid resuscitation. [ 13 ]
A treatment used sometimes is endoscopic band ligation. [27] In 2010, a team of Japanese surgeons performed a "novel endoscopic ablation of gastric antral vascular ectasia". [10] The experimental procedure resulted in "no complications". [10] Relapse is possible, even after treatment by argon plasma coagulation and progesterone. [21]