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Soman was the third of the so-called G-series nerve agents to be discovered along with GA (tabun), GB (sarin), and GF (cyclosarin). When pure, soman is a volatile, corrosive, and colorless liquid with a faint odor like that of mothballs or rotten fruit. [3] More commonly, it is a yellow to brown color and has a strong odor described as similar ...
TCA overdose may occur by accident or purposefully in an attempt to cause death. [2] The toxic dose depends on the specific TCA. [2] Most are non-toxic at less than 5 mg/kg except for desipramine, nortriptyline, and trimipramine, which are generally non-toxic at less than 2.5 mg/kg. [5] [2] In small children one or two pills can be fatal. [6]
Poisoning by a nerve agent leads to constriction of pupils, profuse salivation, convulsions, and involuntary urination and defecation, with the first symptoms appearing in seconds after exposure. Death by asphyxiation or cardiac arrest may follow in minutes due to the loss of the body's control over respiratory and other muscles.
Motor skills: Aging causes decreased physical abilities, such as gross and fine motor skills and reflexes, thereby rendering the driver physically unable to perform at a safe level. [ 21 ] [ 22 ] As age increases, there is a reduction in muscle mass and elasticity, bone mass, central and peripheral nerve fibers. [ 23 ]
Confusing. Trivial. Useless. Laughable. Inconsistent. The road rage over DMV test questions continues for drivers 70 and older.
This causes disturbances across the cholinergic synapses and can only be reactivated very slowly, if at all. Paraoxonase 1 is a key enzyme involved in organophosphate toxicity and has been found to be critical in determining an organism's sensitivity to organophosphate exposure. [26]
Developmental toxicity is any developmental malformation that is caused by the toxicity of a chemical or pathogen. It is the structural or functional alteration, reversible or irreversible, which interferes with homeostasis , normal growth , differentiation , development or behavior.
Lead is a potent neurotoxin whose toxicity has been recognized for at least thousands of years. [97] Though neurotoxic effects for lead are found in both adults and young children, the developing brain is particularly susceptible to lead-induced harm, effects which can include apoptosis and excitotoxicity. [97]