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  2. 3-Methylcrotonyl-CoA carboxylase deficiency - Wikipedia

    en.wikipedia.org/wiki/3-Methylcrotonyl-CoA...

    Carnitine deficiency is found in about 50% of cases. [18] Over 90% of those diagnosed with 3-Methylcrotonyl-CoA carboxylase deficiency by newborn screening remain asymptomatic. The medical abnormalities that present in the few who do show symptoms are not always clearly related to 3-Methylcrotonyl-CoA carboxylase deficiency. [5]

  3. Conjugated linoleic acid - Wikipedia

    en.wikipedia.org/wiki/Conjugated_linoleic_acid

    Non-ruminants, including humans, produce certain isomers of CLA from trans isomers of oleic acid, such as vaccenic acid, which is converted to CLA by delta-9-desaturase. [ 7 ] [ 8 ] In healthy humans, CLA and the related conjugated linole n ic acid (CLNA) isomers are bioconverted from linoleic acid and alpha-linolenic acid , respectively ...

  4. Carnitine-acylcarnitine translocase - Wikipedia

    en.wikipedia.org/wiki/Carnitine-acylcarnitine...

    This disorder disrupts the carnitine shuttle system from moving fatty acids across the mitochondrial membrane, leading to a decrease in fatty acid catabolism. The result is an accumulation of fatty acid within muscles and liver, decreased tolerance to long term exercise, inability to fast for more than a few hours, muscle weakness and wasting ...

  5. Carnitine-acylcarnitine translocase deficiency - Wikipedia

    en.wikipedia.org/wiki/Carnitine-acylcarnitine...

    Carnitine-acylcarnitine translocase deficiency has an autosomal recessive pattern of inheritance. Carnitine-acylcarnitine translocase deficiency is a rare, autosomal recessive metabolic disorder that prevents the body from converting long-chain fatty acids into energy, particularly during periods without food. [ 1 ]

  6. Carnitine biosynthesis - Wikipedia

    en.wikipedia.org/wiki/Carnitine_biosynthesis

    The second step of L-carnitine biosynthesis requires the 3-hydroxy-N ε-trimethyllysine aldolase enzyme. 3-hydroxy-N ε-trimethyllysine aldolase is a pyridoxal phosphate dependent aldolase, and it catalyses the cleavage of 3-hydroxy-N ε-trimethyllysine into 4-N-trimethylaminobutyraldehyde and glycine.

  7. Medium-chain acyl-coenzyme A dehydrogenase deficiency

    en.wikipedia.org/wiki/Medium-chain_acyl-coenzyme...

    Secondary carnitine deficiency is sometimes seen with MCADD, and in these cases, acylcarnitine profiles may not be informative. [3] Urine organic acid analysis by gas chromatography-mass spectrometry (GC-MS) will show a pattern of dicarboxylic aciduria with low levels of ketones. Traces of acylglycine species may also be detected.

  8. Carnitine O-octanoyltransferase - Wikipedia

    en.wikipedia.org/wiki/Carnitine_O-octanoyl...

    Carnitine O-octanoyltransferase (CROT or COT) is a member of the transferase family, more specifically a carnitine acyltransferase, a type of enzyme which catalyzes the transfer of acyl groups from acyl-CoAs to carnitine, generating CoA and an acyl-carnitine. (EC 2.3.1.137) Specifically, CROT catalyzes the chemical reaction:

  9. Palmitoylcarnitine - Wikipedia

    en.wikipedia.org/wiki/Palmitoylcarnitine

    Palmitoylcarnitine is an ester derivative of carnitine involved in the metabolism of fatty acids.During the tricarboxylic acid cycle (TCA), fatty acids undergo a process known as β-oxidation to produce energy in the form of ATP. β-oxidation occurs primarily within mitochondria, however the mitochondrial membrane prevents the entry of long chain fatty acids (>C10), so the conversion of fatty ...