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Vitiligo (/ ˌ v ɪ t ɪ ˈ l aɪ ɡ oʊ /, vi-ti-leye-goh) is a chronic autoimmune disorder that causes patches of skin to lose pigment or color. [1] The cause of vitiligo is unknown, but it may be related to immune system changes, genetic factors, stress, or sun exposure.
The Vitiligo Research Foundation was founded in 2010 by Russian entrepreneur Dmitry Aksenov, whose daughter has vitiligo, after he concluded that there was a lack of research into the disease. [1] It is managed by a small team of permanent staff, [ citation needed ] and led by a Board of Directors (Torello Lotti, Professor, Dermatology Division ...
Hypopigmentation can be caused by hereditary conditions such as vitiligo, melasma, pityriasis versicolor, pityriasis alba, albinism, and fungal infections. [ 4 ] Hyperpigmentation results from an increase in melanin synthesis, which is mostly brought on by sun exposure, dermatological disorders, hormones, aging, genetic factors, skin injuries ...
The convalescent phase is characterized by gradual tissue depigmentation of skin with vitiligo and poliosis, sometimes with nummular depigmented scars, as well as alopecia and diffuse fundus depigmentation resulting in a classic orange-red discoloration ("sunset glow fundus" [5] [8] [7]) and retinal pigment epithelium clumping and/or migration.
Autoinflammatory syndromes are a group of inherited disorders characterized by bouts of inflammatory skin lesions and periodic fevers. [29] [30] Blau syndrome Blau syndrome; Chronic infantile neurologic cutaneous and articular syndrome; Familial cold urticaria (familial cold autoinflammatory syndrome) Familial Mediterranean fever; Hyper-IgD ...
The first World Vitiligo Day (also defined as “Vitiligo Awareness Day” or “Vitiligo Purple Fun day”, from the color chosen as Vitiligo Awareness Color [2]) was observed on June 25, 2011. The choice of June 25 as World Vitiligo Day is a memorial to musical artist Michael Jackson , who suffered from vitiligo from the early 1980s until his ...
The restoration of pigment may be aided by topical application of 0.1% 8-methoxypsoralen, 0.5–1% coal tar, or anthralin, followed by sun exposure. [9] With good outcomes, different topical photochemotherapy regimens (topical psoralen UVA; PUVA) have been utilized to treat postinflammatory hypopigmentation brought on by a variety of illnesses. [2]
Poliosis is present in half of patients with segmental vitiligo. [ 5 ] Vogt-Koyanagi-Harada Syndrome (VKH): VKH is a systemic autoimmune disorder affecting melanin-containing tissues, leading to uveitis , meningitis , and poliosis, which often involves the eyebrows and eyelashes.