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  2. Wallerian degeneration - Wikipedia

    en.wikipedia.org/wiki/Wallerian_degeneration

    Wallerian degeneration occurs after axonal injury in both the peripheral nervous system (PNS) and central nervous system (CNS). It occurs in the section of the axon distal to the site of injury and usually begins within 24–36 hours of a lesion. Prior to degeneration, the distal section of the axon tends to remain electrically excitable.

  3. Nerve injury classification - Wikipedia

    en.wikipedia.org/wiki/Nerve_Injury_Classification

    Axon, endo-, peri-, and epineurium transected. Neurotmesis may be partial or complete. Other characteristics: distal Wallerian degeneration; partial or complete connective tissue lesion; severe sensory-motor problems and autonomic function defect; nerve conduction distal to the site of injury absent (3 to 4 days after lesion)

  4. Diffuse axonal injury - Wikipedia

    en.wikipedia.org/wiki/Diffuse_axonal_injury

    When the axon is torn, Wallerian degeneration, in which the part of the axon distal to the break degrades, takes place within one to two days after injury. [26] The axolemma disintegrates, [ 26 ] myelin breaks down and begins to detach from the cell in an anterograde direction (from the body of the cell toward the end of the axon), [ 27 ] and ...

  5. Neuroregeneration - Wikipedia

    en.wikipedia.org/wiki/Neuroregeneration

    Human axon growth rates can reach 2 mm/day in small nerves and 5 mm/day in large nerves. [4] The distal segment, however, experiences Wallerian degeneration within hours of the injury; the axons and myelin degenerate, but the endoneurium remains. In the later stages of regeneration the remaining endoneurial tube directs axon growth back to the ...

  6. Nerve injury - Wikipedia

    en.wikipedia.org/wiki/Nerve_injury

    During Wallerian degeneration Schwann cells and macrophages interact to remove debris, specifically myelin and the damaged axon, from the distal injury site. [2] Calcium has a role in the degeneration of the damage axon.

  7. Axonotmesis - Wikipedia

    en.wikipedia.org/wiki/Axonotmesis

    Motor and sensory functions distal to the point of injury are completely lost over time leading to Wallerian degeneration due to ischemia, or loss of blood supply. Axonotmesis is usually the result of a more severe crush or contusion than neurapraxia. [1] Axonotmesis mainly follows a stretch injury.

  8. Alcoholic polyneuropathy - Wikipedia

    en.wikipedia.org/wiki/Alcoholic_polyneuropathy

    Alcoholic polyneuropathy is a neurological disorder in which peripheral nerves throughout the body malfunction simultaneously.It is defined by axonal degeneration in neurons of both the sensory and motor systems and initially occurs at the distal ends of the longest axons in the body.

  9. Polyneuropathy - Wikipedia

    en.wikipedia.org/wiki/Polyneuropathy

    The most distal portions of axons are usually the first to degenerate, and axonal atrophy advances slowly toward the nerve's cell body. However, if the cause is removed, then regeneration is possible, although the prognosis depends on the duration and severity of the original stimulus [ medical citation needed ] .